Intro & Caries - PD

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Created by:

AlexisDDS  on February 16, 2012

Subjects:

Preventative Dentistry

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Updated 4/11/12

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Intro & Caries - PD

Head Start (1965)
Program provides dental services to preschoolers with F. need
Allows kids to enter school on equal health basis.
1/74
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Head Start (1965) Program provides dental services to preschoolers with F. need
Allows kids to enter school on equal health basis.
Medicaid 65+ to those with F. need. Insurance protection.
Forms of Dntl Health service (3) 1. by Dr. & Dntl Pros
2. non-govt Dr. & Dntl Pros, partly funded by gov't
3. Dr & Dntl Pros employed by govt (Ex: Military)
Each stage of prevention has examples of (3): Professional
Community
Individual
3 Levels of Preventative Dentistry Primary (prevents)-Motivate individual!
Secondary (prevents progress)
Tertiary (replacement; prevent loss of function)
1ary prevention community fluorinated H2O supply
school fluoridation
Fluo. supplement/mouthrinse programs
school sealants
Fluo. varnishes
1ary prev. Professional sealants
apply topical fluoride
diet council
plaque cntrl programs
dental caries activity test
1ary prev. individual Make Dr. Appt
OHI
Fluo. dentifrices
self applied topical Fluo. products
2ary prev. Pro perio scaling
3ary prev. Pro prosthetics
Educational Process 1. Know Pt. Needs
2. Get Pt. to express needs
3. Provide Pt. Motivation
4. Help Pt. set goals!
5. Activate goals
6. Repeat! -demo, -(Q)s, -different words
7. Evaluation
dental plaque sticky layer of microorganisms/bacteria embedded in matrix.
Begin @ cervical area & interproximally.
May cause disease after 24 hrs.
Detersive foods apples, toast=Deter Plaque!
Which Arch collects more plaque? Mandibular!
(think of salivary glands, plus gravity lets food rest on bottom)
How to detect plaque: direct vision
Explorer/Probe
Disclosing Solutions
Why does lease amount of plaque collect on Lingual Maxillary teeth? Occlusal & Incisal 3rds also retain less plaque!
1ary etiological factor in dental disease? dental plaque
Acid removes what in tooth tissue? minerals
cariogenic produce cavities
decalcify remove minerals
Sugars + Cariogenic Plaque = ACID formula
Acid + tooth surface = CARIES formula
Fermentable Carbs(Carcinogenic): plaque>
bateria>
acid formulation>
pH decrease>
demineralization!
Contributing factors to caries production: 1. time starts immediately
2. pH of plaque
3. frequency of carb intake
4. tooth morphology (alignment)
5. saliva content
6. nutrition/diet
7. systemic factors
8. family habits
9. heredity
Is it better to have an Airhead or a Lollipop? airhead-chew & get it out!
pH level drops with every lick of a pop-no homeostasis
pH decreases after... sucrose is in contact! 6.2-7.0 avg pH range
pH decreases in caries susceptible pt.s to what pH & will increase in how many hours? pH 5.0; 2-3 hours
pH enamel demineralizes... @ pH 5.0
incipient caries lil white spots-demineralized. Fluoride Tx will pause progress
protein helps to ... neutralize acid environment
cariostatic inhibits caries
Health Triangle Physical
Social, Mental
fat protective oily film inhibits acid from getting to tooth structure
(cariostatic; this is How protein helps)
Plaque mineralization change from soft plaque to hard calculus deposit. Occurs quickly after plaque matures.
Avg Calculus formation= 10-20 days
Mineralized plaque/Calculus: 1. acquired pellicle formation (very 1st layer in plaque)
2. plaque formation
3. plaque mineralized/calculus
Which areas of which teeth are more apt to calculus? (b/c salivary glands 1st hit these areas) Buccal of 1st molars,
Lingual of Mandibular anteriors
tissue follows Bone!
periodontitis bone disease
gingivitis inflamed gums disease
ANUG aka "trench mouth" (WWI) Acute Necrotizing Ulcerative Gingivitis- too much bacteria @ interdental papilla.
Quick onset, eats away @ gums, causes ulcers (that harbor plaque & food debris), swelling & dead oral tissues. Pseudomembrane!
Chronic- long onset
Pseudomembrane A false membrane that sometimes develops during infections-looks like real tissue but is usually mucus, fibrin, bacteria or immune system cells.
Systemic Diseases: hormone imbalances
pregnant gingivitis
leukemia
Ascorpic acid deficiency (lack of Vit. C-scurvy)
Gingival Hyperplasia-dylantin
Acute herpetic gingivostomatitis
Gingival Hyperplasia Overgrowth of the Gum tissue, often in pt.s treated with Dylantin (controls epileptic seizures).
Acute herpetic gingivostomatitis =gingivitis + stomatitis. Inflammed gingiva & oral mucosa.
Saliva= source of minerals in SUPRAgingival calculus.
Gingival sulcus fluid= source of minerals in SUBgingival calculus.
Calculus attaches to exposed Cementum (3): 1. acquired pellicle
2. irregularities
3. direct contact
Traits of inflamed gums: change in color, form, position
surface of healthy gingiva="rete-peg!"
when plaque produces toxins... they irritate sulcar epithelium (lining mucosa) which causes gingivitis!
alveolar crest- most coronal part of alveolar bone-interproximal.
alveolus- tooth socket
apposition- formation new layer of tissue/structure
resorption- destruction; loss of tissue/structure.
Cortical bone- aka Compact bone!
Dense; on Facial, Lingual surfaces of mandible & Maxilla.
On Xrays appear very white! Outer Layer of Bone, where needed for strength!
Cancellous bone- Most Interior of Max & Mand.
Lightweight, spongey bone.
On Xrays appear Web-Like! "Trabeculae"
Periodontium Unit= gingiva, periodontal ligaments, cementum, alveolar bone.
5 periodontal ligaments Alveolar Crest
Horizontal
Oblique
Apical
Interradicular
White Lamina Dura- healthy (on xray) Lines alveolus
material alba lots of plaque (becomes mature)
periodontal ligament specialized CT surrounds root & connects it to Alveolar Bone.
Plaque begins where? @ Cervical areas & proximal surfaces
Distribution considerations-plaque formation crowding, hygiene, prosthetics, supragingival, subgingival
Dry Socket AKA Alveolitis!
inflammation & Infection associated w/disturbance of blood clot after EXT!
Clot is important b/c protects wound & is later replaced by granulation tissue, then bone!
Curettage removing bacteria from pocket/lining of tooth! (soft tissue)
Root Planning remove bacteria on cementum of tooth! (hard tissue)
gingivitis is reversible
Periodontitis is irreversible
Plaque: 1.Acquired Pellicle
2.Bacteria collects
3.Plaque matures!
rete pegs: fibers from bone to tissue. When healthy it is taunt, tight and stippled.
Loss of rete pegs="loss of stippling"=1st step of gingivitis.
bulbous/swollen tissue area indicates advanced gingivitis!
shiny, smooth, red, bulbous tissue means Bacteria is underneath gingiva! No rete pegs, just smooth!
Pseudopocket pocket of swollen tissue (measurement appears larger because of swelling)
Pseudopocketing means swollen, not bone loss!
It's only a true pocket if ... if bone is lost!

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