sensory physi
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jturner916 on March 2, 2012
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52 terms
Terms | Definitions |
|---|---|
Sensory Function | specialized cells that monitor internal and external conditions |
Two types of senses | ... |
General | receptors are distributed throughout the body1. Afferent impulses are sent to the somatosensory cortex |
Special | receptors are concentrated in sense organs1. Afferent impulses are sent to special sense cortexes |
Afferent (sensory) division | carries sensory info from receptors to the CNS |
i. Somatic afferent fibers | carries impulses from skin, skeletalmuscles and joints |
ii. Visceral afferent fibers | carries impulses from organs withinventral body cavities |
iii. Special sense afferent fibers | eyes, ears, taste, smell |
Pseudounipolar neuron(sensory neuron) except special sensory | 1. Cell body lies in the dorsal root ganglia2. Action potential is initiated at the peripheral end of the neuron |
Sensory Receptors | -found on the peripheral end of sensory neurons. -Act as transducers to change an incoming stimulus of one type into an electrical impulse |
Adequate stimulus | receptors are specific for the type of stimulus it issensitive to an can transducer, ie cants shine light in ear and hear something. |
...exception | Receptors can respond to other types of stimuli if strong enough. ie press on eye (mechainal stress) you will see flashes of light. |
"Law of Specific Nerve Energies | regardless of the type of stimulus the sensation is perceived as what the receptor is specific for |
Sensory Transduction | i. A stimulus alters the membrane permeability of the receptor. This leads to the production of a graded receptor potential |
Sensory Transduction | ii. Receptors that are modified endings of the afferent neuron produce generator potentials |
Sensory Transduction | iii. Receptors that are separate cells from the afferent neuron produce receptor potentials |
Sensory Transduction | iv. Release chemicals that open ligand gated channels on the afferent neuron |
i. Free nerve endings | 1. Nociceptors2. Themoreceptors |
1. Nociceptors | activation causes the sensation of paina. Free nerve endings found in the skin, joints, bones, and blood vessels b. Sensitive to chemicals, tissue damage, and extreme temperatures |
2. Themoreceptors | react to changes in temperaturea. Free nerve endings in the dermis, hypothalamus, and liver b. You have 3X more cold receptors than warm receptors, cold vs colder= more action potiental high fequency, 10 degree or less you feel pain /45 then pain |
Mechanoreceptors | stimulated by physical change such as pressure or movement |
Merkel cells | cells in the stratum basale associated withfree nerve endings, detect fine touch |
Hair root plexus | free nerve endings associated with hair follicles, detect movement of hair |
Meissner's corpuscles | -detect touch, found in dermal papillae |
Ruffini corpuscles | sensitive to stretch and distortion, found in the dermis |
Pacinian corpuscles | deep pressure receptors found in the dermis |
Baroreceptors | sensitive to internal pressures. Monitorsblood pressure in vessels (carotid sinus and aorta), lungs, bladder, intestines |
Proprioceptors | monitors position and stretch of muscles, tendons, ligaments, and joint capsules. Helps maintain posture and sense of body position |
Chemoreceptors | detect chemicals dissolved in solution. Olfactory, taste, osmolarity, pH, CO2, O2 alot of special senses |
Ascending tracts | a bundle of axons transmitting impulses towards thebrain. Usually involves three successive neurons (three levels of processing) tend to be on dorsal and lateral sides of your spinal cord |
i. Receptor level | Primary neuron running from the receptor to theposterior horn of the spinal cord or medullary nuclei |
ii. Circuit level | Secondary neuron synapses with the first and transmit impulses to the thalamus (all sensors go to thalamus but olfactory) |
iii. Perception level | Tertiary neuron running from the thalamus tothe primary somatosensory cortex or a special senses cortex. The cerebral cortex is responsible for |
cerebral cortex is responsible for | 1. Sensation - the awareness of the stimuli2. Perception - the interpretation of the stimuli |
Each sensory fiber is a "labeled line | the type, location, strength, and the intensity of the stimuli is encoded in the area of the cortex it travels to and the frequency of the impulse |
Determining type of stimulus or receptor activated | 1. Depends on the pathway it takes and the area of thecerebral cortex it travels to |
Determining type of stimulus or receptor activated | 2. The area of the cortex devoted to each region is related to the regions sensitivity (# of receptors |
Referred pain | many primary neruons sinape with the same secondary neruon, usually defalt to the most frequently stimulated ie skin not heart |
Phantom pain | remodeling of the cortex, or a secondary neuron is irritated |
Localization of the stimulus | Accuracy of localization depends on the size of thereceptor field. The smaller the receptor field the more accurate. |
Localization of the stimulus | Accuracy also depends on lateral inhibition. Only themost intensely stimulated pathway is excited. Less excited pathways surrounding the stimulated area are inhibited |
Number of receptors stimulated - stronger stimuliusually affect larger areas | Number of receptors stimulated - stronger stimuliusually affect larger areas |
usually affect larger areas | Frequency of action potentials - stronger stimuli generatelarger receptor potential, therefore a greater frequency of action potentials |
Duration of the stimulus | Adaptation - receptors change their sensitivity in thepresence of constant stimulus ie a watch on your wrist |
Phasic receptors | fast adaptingi. Report changes in the environment ii. Burst of firing at the beginning and end of stimulus iii. Examples: temperature, smell, touch |
Tonic receptors | slow adapting or not at alli. Constant firing rate ii. For situations were continuous information about a stimulus is valuable iii. Examples: proprioceptors pain, muscle stretch |
pain | is subjective , influenced by past adn present experiences, and is coupled with emotional responses |
fast pain pathways | sharp pain, easy to localize, myleineated |
slow pain pahtways | unmyleined, dull aching pain, persists long and hard to locialize |
what stimulates the pain pathway | nociceptors are sensite to chemcials released by damaged tissues. prostaglandins, bradykinins, histamine, capsaicin |
pain pathway | nocicieptors stimulated and primary neurion relases suspstance P- secondary neuron then thalamus and to the reticular formation(brain stem- level of alertness) and hypothamlus( emotinal response) |
analgesic pathway | endorphins bind to opiate recepotros on the axon termainal and block the release of susbstance P(example of presnapic inhibition) |
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