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7 Written questions

6 Multiple choice questions

  1. -decrease production of neurotransmitters that transmit pain
  2. -change in pain etiology from tissue to physiologic impair-
    ment including muscle guarding, abnormal movement, disuse, decreased endurance of back/abdominal muscles;
    more area of pain matrix are activated and malfunctioning;
    can also see depression, sleep disturbance, preoccupat.
    with pain decreases activity, fatigue, emotional distress
  3. - (Sypatheric Reflex Dystrophy) abnormal response to trauma leading to limb disuse; disuse of limb precipitates an increase in levels of neurochemicals that cause peripheral inflammation, along with a decrease in sympathetic regulation of blood flow &swating; symptoms include pain, vascula changes, atrophy affecting entire hand/foot, red/pale skin, excessive sweating, edema, late stage dry/cold skin, muscle atrophy and osteoporosis.
  4. -cutting of a dorsal sensory nerve root
  5. -If peripheral sensory information is completely absent, CNS neurons in the nociceptive pathway become abnormally active
  6. -increases circulation th compression of tissue which decreases ischemic pain, a deficiency of blood (ischemia) results in a deficiency of 02 (hypoxia) which cause pain in tissue; pain from cramp is due to squeezing of capillaries & decreasing blood supply; when blood flow is interrupted,
    pain-stimulating chemicals accumulate; applied heat dilates blood vessels, increasing blood flow/02 and decrea
    sing the concentration of pain-stimulating substances; massage also simulates A-beta fibers thus activating the gating contrl mechanism

6 True/False questions

  1. Sympathectamy- partial excision of synpathetic nerves or ganglia

          

  2. Phantom Limb Pain-post amputation, absence of sensory info causes neurons in central nociceptive pathways to become overactive; maladaptive structural reorganization is found in the SC,
    thalamus, and cerebral cortex; there is overlap of cortical reprsentation that are normally separate (loss of sensory info lighting up pain matrix)

          

  3. Deafferentation-tenderness/stiffness of muscles, aching pain, increased stress/sllep disorders, secondary hyperalsia, allodynia; abnormal activation of pain matrix with amplification of signals

          

  4. 1) PNS-If peripheral sensory information is completely absent, CNS neurons in the nociceptive pathway become abnormally active

          

  5. Mononeuropathy-involve more than one nerve, such as diabetic neuropathy and Guillian Barre

          

  6. Peripheral-Aspirin/ibuprofin decreases synthesis of prostaglandins
    preventing prostaglandins from sensitizing nociceptors.

          

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