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Mediastinal Lymphadenopathy

Hodgkin's Lymphoma

EBV association

Hodgkin's Lymphoma

Nodular Sclerosing

most common type of Hodgkin's Lymphoma
(lymphocyte predominant)

t(8;14)

Burkitt's Lymphoma

t(11;14)

Mantle Cell Lymphoma

t(14;18)

Follicular Lymphoma

c-myc activation

Burkitt's Lymphoma

cyclin D deactivation

Mantle Cell Lymphoma

bcl-2 expression

Follicular Lymphoma

Most common adult non-Hodgkin lymphoma

Diffuse Large B-Cell Lymphoma

HTLV-1

Adult T-Cell Lymphoma

CD4+

Mycosis Fungoides/Sezary Syndrome

monoclonal IgM spike

Waldenstrom's macroglubulinemia
(not Multiple Myeloma!)
it causes hyperviscosity, not bone problems

Rouleaux formation

Multiple Myeloma

"Fried Egg" cell on light microscopy

Plasma cell in Multiple Myeloma

"Clockface Chromatin"

Multiple Myeloma

t(12;21)

if found in ALL, signifies a BETTER prognosis

CALLA+

ALL

warm antibody autoimmune hemolytic anemia

CLL/SLL (chronic/small)

tartrate-resistant acid phosphatase

Hairy Cell Leukemia

t(15;17)

AML (M3 subtype) aka Acute Promyelocytic Leukemia

Auer Rods

AML
Peroxidase-positive inclusions in granulocytes and myeloblasts. Treatment Drugs can cause rupture cells and release peroxidase, causing DIC!

Treated with all-trans retinoic acid (Vitamin A)

AML

Presents with DIC

AML

t(9;22)

CML

bcr-abl

CML

Molecular Signaling Mechanisms of...
c-myc
bcl-2
cyclin D
bcr-abl

look it up!

LOW leukocyte alkaline phosphatase

CML

"Blast Crisis"

CML turns into AML or ALL

t(11;22)

Ewing's Sarcoma
(non-lymph)

Age > 60 years

Chronic = CLL
Acute = AML

Age < 60 years

Chronic = CML
Acute = AML
(or hair cell possible)

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