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5 Written Questions

5 Matching Questions

  1. Clinicopatholgic correlations of shock
  2. Pathology of shock
  3. Edema
  4. Paradoxical emboli
  5. 4 Arterial Thrombi outcome
  1. a emboli that travel from venous arterial circulation via a communication between arterial and venous circulation
  2. b 1. resolution
    2. organization/incorporation
    3. embolization (arterial)
    4. propagation
    *all similar to venous thrombi
  3. c excess fluid in the interstital spaces of the body
  4. d -early of compensated shock
    -decompensated, but reversible shock
    -irreversible shock
  5. e (upon autopsy) interal organs are congested and wet from edema; the lungs are 2-3 times heavier. the liver is congested and blood oozes from it. the intestines are dark from blood pooling and wet. the kidneys are swollen, pale, and congested. and the brain is edematous, flattened gyri.

5 Multiple Choice Questions

  1. loss of blood volume; i.e. hemorrhages, water loss from burns, vomitting/diarrhea
  2. loss of peripheral vascular tone and pooling of blood in dilated peripheral blood vessels
  3. a detached solid, gaseous, or liquid mass that is carried by the blood from site of origin to a distal site. May lodge in pulmonary or systemic circulation.
  4. mixed acidosis occurs. result of renal, anaerobic glycolosis, and respiratory insufficiency.
  5. constriction of the renal cortical vessels reduces GFR. results in renal output.

5 True/False Questions

  1. Red infarcthemorrhagic; organs with dual blood supply; soft aerated tissues

          

  2. Pumonary Infarctemobilzation to small distal vessels in lung may cause ischemic necrosis of tissue infarct

          

  3. Is shock reversible?early stages of shock are reversible and treatable. in the early stages, peripheral vasoconstriction helps compensate for hypoperfusion. the central pooling redirects blood to vital organs. if untreated, can cause death.

          

  4. Compensated shocka set of adaptations occur to compensate for the circulatory imbalance. i.e. tachycardia, vasoconstriction of peripheral arteries, and reduced urine production

          

  5. Cardiogenic shockpump failure; i.e. secondary to MI, conduction block or arrythmia, myocarditis or valvular heart disease

          

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