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5 Written Questions

5 Matching Questions

  1. Oliguria
  2. Sequelae
  3. Decompensated shock
  4. Clinicopatholgic correlations of shock
  5. Cardiogenic shock
  1. a pump failure; i.e. secondary to MI, conduction block or arrythmia, myocarditis or valvular heart disease
  2. b when early shock fails: hypotension occurs, BP and CO fall. Tachypnea and SOB lead to heart failure and pulmonary edema causing anoxia, lead to ARDS.
  3. c sudeen death, clinically silent; resolution, organization, dyspnea, pulmonary infarct
  4. d -early of compensated shock
    -decompensated, but reversible shock
    -irreversible shock
  5. e constriction of the renal cortical vessels reduces GFR. results in renal output.

5 Multiple Choice Questions

  1. loss of peripheral vascular tone and pooling of blood in dilated peripheral blood vessels
  2. major factors PUSHING fluid out of vessel
  3. emobilzation to small distal vessels in lung may cause ischemic necrosis of tissue infarct
  4. the end result of decompensated shock. marked by: circulatory collapse, hypoperfusion of vital organs, and loss of vital functions. PTs are in distress and are frantic, DIC is common, and high mortality.
  5. Thrombi, fat, amniotic fluid, foreign substances, and bone marrow

5 True/False Questions

  1. Patho of Pulmonary Thromboembolism-20 to 25 per 100,000 PTs have it
    -fatal if 60% of circulation is obstructed

          

  2. Compensated shocka set of adaptations occur to compensate for the circulatory imbalance. i.e. tachycardia, vasoconstriction of peripheral arteries, and reduced urine production

          

  3. Pulmonary Thromboembolism-20 to 25 per 100,000 PTs have it
    -fatal if 60% of circulation is obstructed

          

  4. Hyperemia (erythema)increased inflow into a vessel; i.e. exercise, inflammation

          

  5. Infarctionhemorrhagic; organs with dual blood supply; soft aerated tissues

          

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