4 Written Questions
4 Multiple Choice Questions
- inflammation of the gray matter of the spinal cord; caused by an Enterovirus that attacks Anterior Horn Cells & sometimes motor nuclei of brainstem, often resulting in spinal and muscle deformity and paralysis (polio = gray); Systemic illness prior to neurologic symptoms
Clinical Manifestation: Muscles become painful & stiff, LMN asymmetrical weakness with prominent atrophy over time; survivors often develop postpolio syndrome
- degeneration of corticospinal tracts, posterior columns, spinocerebellar tract caused by the inability to absorb vitamin B12
Clinical presentation: Loss of proprioception, vibratory sense, light touch, motor
- 1- Syrinx (fluid-filled, slit-like cavity) develops in the spinal cord or brain stem (syringobulbia), causing damage to the crossing fibers of spinothalamic tract and resulting in neurological disability. Most secondary to trauma or congenital hydrocephalus (Arnold-Chiari Malformation).
2- Clinical manifestations include wasting of intrinsic muscles of hand, spastic weakness in legsshawl like, bilateral loss of pain and temperature sensation in the upper extremities with preservation of touch and sensation.
- brain inflammation caused by a virus
4 True/False Questions
Cervical spondylosis → degenerative condition of intervertebral discs in cervical region. Associated w/ degeneration of facet jts, hypertrophy of ligamentum flavum, and ossification of longitudinal ligament and bone spurring leading to spinal cord, nerve root and vertebral artery compression
Amyotrophic Lateral Sclerosis → degenerative condition of intervertebral discs in cervical region. Associated w/ degeneration of facet jts, hypertrophy of ligamentum flavum, and ossification of longitudinal ligament and bone spurring leading to spinal cord, nerve root and vertebral artery compression
Friedreich's Ataxia → progressive degeneration of spinocerebellar and corticospinal tracts and dorsal columns; genetic cause-autosomal recessive disorder
Caisson disease → Decompression sickness; occlusion of epidural venous sinuses by nitrogen bubbles forced out of solution by rapid decompression; impaired venous return and hemorrhagic infarcts in spinal cord affecting the thoracic region first
Clinical Presentation: joint pain, skin rash, edema, parasthesias and paralysis in LE more than UE