In certain large animals, this type of neuron can extend beyond one meter in length
a sensory neuron.
For a neuron with an initial membrane potential at -70 mV, an increase in the movement of potassium ions out of that neuron's cytoplasm would result in
hyperpolarization of the neuron.
Though the membrane of a "resting" neuron is highly permeable to potassium ions, its membrane potential does not exactly match the equilibrium potential for potassium because the neuronal membrane is
slightly permeable to sodium ions.
The operation of the sodium-potassium "pump" moves
sodium ions out of the cell and potassium ions into the cell.
A "resting" motor neuron is expected to
exhibit a resting potential that is more negative than the "threshold" potential.
The "threshold" potential of a membrane
is the minimum depolarization needed to operate the voltage-gated sodium and potassium channels.
A toxin that binds specifically to voltage-gated sodium channels in axons would be expected to
prevent the depolarization phase of the action potential.
After the depolarization phase of an action potential, the resting potential is restored by
the opening of voltage-gated potassium channels and the closing of sodium activation gates.
In the sequence of permeability changes for a complete action potential, the first of these events that occurs is
the opening of voltage-gated sodium channels.
Saltatory conduction is a term applied to conduction of impulses
jumping from one node of Ranvier to the next in a myelinated neuron.
Neural transmission across a mammalian synaptic gap is accomplished by
impulses causing the release of a chemical signal and its diffusion across the gap.
The observation that the acetylcholine released into the junction between a motor neuron and
a ligand-gated sodium channel.
An inhibitory postsynaptic potential (IPSP) occurs in a membrane made more permeable to
The activity of acetylcholine in a synapse is terminated by
its degradation by a hydrolytic enzyme on the postsynaptic membrane.
When several EPSPs arrive at the axon hillock from different dendritic locations,
When several IPSPs arrive at the axon hillock rapidly in sequence from a single dendritic location, hyperpolarizing the postsynaptic cell more and more and thus preventing an action potential, this is an example of
Assume that a single IPSP has a negative magnitude of - 0.5 mV at the axon hillock, and that a single EPSP has a positive magnitude of + 0.5 mV. For a neuron with initial membrane
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potential is -70 mV, the net effect of the simultaneous arrival of 6 IPSPs and 2 EPSPs would be to move the membrane potential to
The primary neurotransmitter from the parasympathetic system that influences its autonomic
What happens when a neuron's membrane depolarizes?
The neuron's membrane voltage becomes more positive.
Why are action potentials usually conducted in only one direction along an axon?
The brief refractory period prevents reopening of voltage-gated Na+ channels.
A common feature of action potentials is that they
are triggered by a depolarization that reaches the threshold.
Which of the following is a direct result of depolarizing the presynaptic membrane of an axon terminal?
Voltage-gated calcium channels in the membrane open.