Unit XI: Nursing Care Child with GI issues

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Created by:

prcooley  on January 4, 2011

Subjects:

nursing

Classes:

West Penn Class of 2011

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Unit XI: Nursing Care Child with GI issues

FLACC
Face, Legs, Activity, Cry and Consolability
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FLACC Face, Legs, Activity, Cry and Consolability
FLACC scale is used for Pain tool for 6 months and older
NPO for solids 8 hours
NPO for formula 6 hours
NPO for breast milk 4 hours
NPO for clear liquids 2 hours
If NPO after midnight, do what after midnight start IV, provide glucose so sugards don't drop, infants may not be able to store much glycogen
Skin turgor pinch inner thighs and belly - check hydration status
Best way to check Hydration Status Weight
increments of measureing fluid loss every 4 hours
chemical pneumonia when milk or breast milk is aspirated due to stickiness
Foregut from pharynx to proximal duodenum, including liver, pancrease, biliary tract and lower respiratory tract
Midgut from distal duodenum to transverse colon
Hindgut from descending colon to anal canal
should be present at birth sucking & swallowing
should be present at birth extrusion reflex
gastric PH in new born 1-3, compared to (0.1-1.5 in adult)
lipase pancreatic enzyme that breaks down lipids/fats
protease (trypsin) pancreatic enzyme that breaks down protein
amylase pancreatic enzyme that breaks down carbs and starch
bile emulsifies fat
by age 2 GI mature, sphincter control (potty training)
trachealesophogeal fistual & esophageal atresia failure of the esophagus to develop as a continuous passageway
fistula opening between two adjacent areas
atresia blind pouch
polyhydraminos is associated with trachealesophageal fistula and esophageal atresia, because amniotic fluid builds up
3 Cs Cough, choke, cyanosis
abdomen fills with air trachealesophageal fistula and esophageal atresia
ferril bag an empy bag that releases gas into it
esophageal strictures narrowing, scaring, stiffness
one anastomosis risk respiratory distress
pyloric stenosis an obstruction, at the outlet of the stomach
pyloric stenosis 5x more likely in males
pyloric stenosis progressive hypertrophy of circular muscle
pyloric stenosis projectile vomiting (2-4 feet)
pyloric stenosis dehydration, weight loss, baby with sunken fontanels
olive shaped mass in RUQ seen in pyloric stenosis (enlarged pyloris)
pyloromyotomy muscle splitting procedure
pyloromyotomy should remain high fowlers 20-30 min.
pyloromyotomy put on slight right side, it facilitates drainage
pyloromyotomy hypoglycemic
gastroesophageal reflux call ruminators, comes back up because of incompetent esophagus
begins at 20 weeks swallowing
gastroesophageal reflux vomiting/spitting
gastroesophageal reflux hiccupping
gastroesophageal reflux recurring otits media
gastroesophageal reflux failure to thrive because losing weight, always hungry
Gold standard for Acid reflux test TUTTLE test
TUTTLE test pH probe, placed in stomach, as multimple sensors, placed in for 24 hours, only apple juice and water are allowed
reglan increases lower esophageal pressure & gastric emptying time
Nissen fundoplication take the fundus of the stomach and wrap it around the lower esophageal spincter, 270 wrap or 360 wrap
can't vomit 360 nissen fundoplication wrap
omphalocele congenital defect in which a sack containing abdominal contents protrudes from the umbilical stump.
omphalocele occurs between 6-10 week, midgut grows, it elongates, and it projects from the abdomen, often returns to normal by 11th week
gastroschisis congenital defect where the abdomen wall has formed but there is a weakness or herniation, usually to the right or left of an intact umbilical cord
gastroschisis can have twisted, leathery or necrotic intestines; circulation can be cut off, higher in males
omphalocele have warm IV saline drip onto gaze
omphalocele & gastroschisis might need to be NPO for several weeks after surgery.
intussuception most common cause of intestinal obstruction from 3 months to 3 years
intussuception can have red, currant jelly-like stools, because blood leaks into the intestines
intussuception felt in lower right quadrant
hirschsprung disease absence of nerve innervations to a portion of the colon
hirschsprung disease constipation, abdominal distention, vomiting
barium enema shows dilated groups of bowel
anal agenesis similar to atresia (did not descend totally)
how to treat anal stenosis periodic dilatations, sitz bath
breastfed stool yellow, pasty, sour milk odor
bottlefed brown stool
extrahepatic biliary atresia can be resolved by making an opening to connect to the liver to drain
intrahepatic biliary atresia will require a liver transplant
jaundice is first seen in the sclera
vitamin issues with biliary problems vit A (vision), D (calcium uptake), E (supposed to help prevent hemolysis), K (clotting)
calcium supplements with obstructed bile flow
binds to fat calcium
gastroenteritis massive diarrhea
gastroenteritis can result in sepsis, shock, or death
gastroenteritis inflammation can result in ulceration, bleeding, sepsis
enterotoxins will cause massive loss of fluids
cytotoxins will cause local edema of the intestine, malabsorption and dehydration
neurotoxins (shigella) attacks the nerve cells
gastroenteritis often seen in daycares, peaks in summer
gastroenteritis can be water borne, salmonella,
metabolic acidosis loss of NA, K, and NaHCO3
tissue hypoxia lactic acid build up
fat break down ketosis
losing 15% of weight very bad, near death
blood pressure drops very last sign (severe sign)
1ml/1kg/1hr minium urinary output for a young child
celiac disease inability to tolerate wheat protein (gluten); symptoms include foul-smelling diarrhea and emaciation; often accompanied by lactose intolerance
celiac disease first evidence may be growth failure and diarrhea
celiac disease when it becomes a crisis, can lead to explosive watery diarrhea
celiac disease abdominal distension
celiac disease mucosal inflammation, crypt hyperplasia, villous atrophy
celiac disease gluten free diet is the cure
celiac disease high calorie, protein, simple carbs, low in fat
lactose intolerance Body is lacking the enzyme (lactase) that breaks apart lactose (glucose+galactose). Then, the molecules are 'too big.' Instead, bacteria come by and eat up the huge lactose molecules, and their digestive byproducts are the cause of the bowel irritation (gas, diarrhea, etc.).
breath hydrogen test diagnose lactose intolerance
lactose intolerance for infants, use fecal pH test (acidic +)
Encopresis involuntary defecation not attributable to physical defects or illness
Encopresis inappropriate, and must be 4 years old
Encopresis constipation can be a cause
steatorrhea greater than normal amounts of fat in the feces which are frothy and foul smelling and floating
cries scale used for less than 6 months

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