Microbiology for Embalmers - LU 6

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Hutch0055  on June 6, 2011

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microbiology for embalmers

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Microbiology for Embalmers - LU 6

Rickettsia
1. Are OBLIGATE INTRACELLULAR PARASITES.
2. They are very similar to bacteria and divide by
BINARY FISSION.
3. They are COCCOBACILLARY in shape.
4. They are transmitted TO MAN VIA INSECTS who are
the natural and primary hosts.
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Rickettsia 1. Are OBLIGATE INTRACELLULAR PARASITES.
2. They are very similar to bacteria and divide by
BINARY FISSION.
3. They are COCCOBACILLARY in shape.
4. They are transmitted TO MAN VIA INSECTS who are
the natural and primary hosts.
Rickettsia - Pathogenicity1. Invade the reticuloendothelial system, colonizing the
endothelial lining of cells of the walls of small blood
vessels (small arteries and capillaries).
2. Vasculitis occurs and is spread all over the body
(disease in many anatomic areas).
3. A skin rash and many pathological changes occur
and this can be life threatening.
4. An attack of ____________ disease is usually followed
by a lasting immunity.
Rickettsia prowazekii 1. The etiological agent of typhus fever or epidemic
typhus.
2. The biological vector is the louse (lice plural).
Rickettsia prowazekii - Pathogenicity1. A louse bites a person with epidemic typhus.
2. The rickettsias are taken into the stomach of the
louse and from there invade the intestinal tract of
the louse.
3. There they multiply and the cells burst causing the
agent to enter the feces.
4. When the louse bites the person it defecates at the
same time.
5. The bite itches the person and the rickettsia goes
into the skin after the person scratches the bite.
6. Severe headache, chills, fever and rash result within
a week.
7. 10-40% mortality
Rickettsia prowazekii - Transmission Bite of the louse.
Rickettsia prowazekii - Entry and Exit ENTERS by way of the bite (of louse).
NO EXIT
Rickettsia prowazekii - Immunity Immunity after attack (from louse bite)
Rickettsia rickettsii 1. Is the etiological agent of rocky mountain spotted
fever.
2. The biological vector is the tick.
Rickettsia rickettsii - Pathogenicity Fever, rash and petecchial hemorrhages.
Rickettsia rickettsii - Transmission Bite of a tick
Rickettsia rickettsii - Entry and Exit ENTERS by way of the bite (of tick).
NO EXIT
Rickettsia typhi 1. The etiological agent of endemic (flea borne) typhus.
2. The biological vector is the flea.
3. The natural infection of rats.
Rickettsia typhi - PathogenicitySimilar to Rickettsia prowazekii.
1. A FELA bites a person with epidemic typhus.
2. The rickettsias are taken into the stomach of the
louse and from there invade the intestinal tract of
the louse.
3. There they multiply and the cells burst causing the
agent to enter the feces.
4. When the louse bites the person it defecates at the
same time.
5. The bite itches the person and the rickettsia goes
into the skin after the person scratches the bite.
6. Severe headache, chills, fever and rash result within
a week.
Rickettsia typhi - Transmission Bite of the flea
Rickettsia typhi - Entry and Exit ENTERS by way of the bite (of Flea).
NO EXIT
Rickettsia typhi - Immunity Immunity after attack (from flea bite)
Coxiella burnettii 1. The etiological agent of Q FEVER.
2. There is NO INSECT VECTOR.
3. This is a ZOONOTIC AGENT involving goats, cattle
and sheep.
4. Occupational hazard for people working around
these animals.
Coxiella burnettii - Pathogenicity 1. This organism does not fit the profile of other
rickettsia.
2. There is fever, no rash but symptoms similar to
atypical pneumonia or influenza.
Coxiella burnetti - Transmission 1. Raw milk of infected cows.
2. Inhalation of contaminated air from dairy barns and
lambing sheds.
Coxiella burnettii - Entry and Exit ENTERS by mouth or nose.
NO EXIT
Coxiella burnettii - Immunity No Immunity for the agent of Q FEVER
Chlamydia trachomatis 1. Is the etiological agent of trachoma,
lymphogranuloma venereum, and non-gonococcal
urethritis (NGU) or non-specific urethritis (NSU).
2. Please note: Chlamydia is another genus of
bacterial-like organisms similar to rickettsia in that
they are OBLIGATE INTRACELLULAR PARASITES.
Trachoma - Pathogenicity 1. Attacks the lining cells of the cornea and
conjunctiva.
2. The scar tissue causes a pebble-like appearance to
conjunctiva.
3. It is the world's leading cause of blindness/visual
disability.
4. It is endemic in underprivileged areas.
Trachoma - Transmission Flies transmit mechanically, fingers and fomites as well.
Trachoma - Entry an Exit ENTERS by way of the eyes.
EXITS by secretions from the eyes.
Trachoma - Immunity No Immunity for this cause of Blindness / Visual Disability
Lymphogranuloma venereum A sexually transmitted similar to gonorrhea as it is ONLY IN HUMANS.
Lymphogranuloma venereum - Pathogenicity1. In men the primary ulceration is on the external
genital organs. The agent travels to the inguinal
lymph nodes and buboes are formed.
2. In women from the primary lesion in the genital
tract it travels to lymph nodes within the pelvis
where the chronic inflammation leads to stricture of
rectum.
3. In both men and women, the infection is mainly in
the lymphatics, deformity results on the outer
genitalia.
4. Fever, chills, headache, joint pains continue during
lymph progression.
Lymphogranuloma venereum - Transmission Primarily direct contact/sexually transmission.
Lymphogranuloma venereum - Immunity No Immunity for this STD which is similar to Gonorrhea
Non-Gonoccocal urethritis - Pathogenicity1. This organism's symptoms closely parallel that of
Neisseria gonnorheae relative to the urethritis,
cervicitis and the conjunctivitis of the new born and
adult.
2. The incubation period of Chlamydia trachomits is
longer and the symptoms are milder.
3. However, the chronic nature of this unchecked
infection is on a par with Neisseria gonnorheae.
Non-Gonococcal urethritis - Transmission 1. Sexual contact.
2. Contaminated hands.
Non-Gonococcal urethritis - Immunity No Immunity for this organism that causes urethritis, cervicitis and conjunctivitis
Chlamydia psittaci 1. The etiological agent of psittacosis and ornithosis.
2. As ZOONOTIC INFECTION involving parrots and
parakeets for psittacosis and domestic fowl and
birds (chicken, turkey, pigeon) for ornithosis.
Chlamydia psittaci - Pathogenicity 1. There is fever, headache, pneumonic involvement
and a heavy cough.
2. Mortality is high.
Chlamydia psittaci - Transmission 1. Man contracts the disease from birds and/or their
infected material by inhalation.
2. Handling sick birds and bites and wounds inflicted
by sick birds.
Chlamydia psittaci - Entry and Exit ENTERS by way of the respiratory tract. (From birds)
NO EXIT
Chlamydia psittaci - Immunity Immunity is unknown for this zoonotic infection from birds

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