5 Written questions
5 Matching questions
- Describe the wind-up theory.
- Where is the cervical sympathetic chain located?
- Describe the dorsal horn changes that occur w/ sympathetically mediated pain.
- In regards to the SNS, where do preganglionic B fibers originate?
- What are the collateral ganglia?
- a In the intermediolateral cell column segments T1-L2 or L3
- b Lies on the anterolateral surface of the vertebral bodies.
- c Involves repetitive noxious stimuli (c-fibers)->prolonged discharge of dorsal horn cells-> progressive ↑ in APs per stimulus->long term potentiation (LTP).
- d A-beta fibers form abnormal connections in shallow laminae (1 & 2)->normal sensory input interpreted as high intensity noxious stimuli.
↑ gene expression for receptors for pain related neurotransmitters (substance P).
↓ opioid binding sites.
- e Cardiac, celiac, superior % inferior mesenteric ganglia.
5 Multiple choice questions
- With the ventral ramus.
- Iris & ciliary body
Lacrimal & salivary glands
- Stomach, small bowel, adrenal medulla
- Paravertebral ganglia T1-L2 on the anterolateral surface of the vertebral bodies.
5 True/False questions
Describe the lumbo-sacral lateral chain → Receives all presynaptic input enters from L1 & L2.
These ganglia are not fused but the coccygeal ganglia meet at the midline
What are the NMDA antagonists, what are they useful for? → Aspartate, glutamate, etc-released by c-fiber stimulation.
Blocking what ganglion will knock out all the sympathetics, why, and what can it result in? → Stellate ganglion. All preganglionic fibers arise below the stellate ganglion.
Could result in Horner's syndrome
What does C fiber sensitization lead to? → Complex regional pain syndrome.
Blocking what plexus is indicated in treatment of intraabdominal malignancies & chronic pancreatitis; diagnostic (local or therapeutic (neurolytic block)? → Celiac plexus