acls

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Created by:

mscandyrn  on June 25, 2011

Description:

Arrythmia's

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acls

WPW
characterized by narrow P waves, 0.8 or less, delta waves, and inverted T-waves
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WPW characterized by narrow P waves, 0.8 or less, delta waves, and inverted T-waves
Atrial Tachycardia Supraventricular tachycardia(SVT)..Rapid regular rhythm 160-200 Beats per minute, When rate ABOVE 160, you know its not coming from SA node...ectopic focus in atria is taking control of rhythm...TX. meds, valsalva maneuver, cardioversion
PSVT regular rapid, narrow QRS complex tachycardia that starts and stops abruptly. (P waves hard to see on ECG) abnormal conduction through a reentry circuit
digoxin toxicity rhythm Like atrial tachycardia with AV block, will see 2 P waves for every QRS. Ventricular rate is half of the atrial rate
Premature atrial contractionsA benign arrythmia that is classified by the presence of premature P waves followed by a normal QRS and T waves. If the impulse is so early that the AV node has not fully repolarized, may not be followed by QRS. The premature P wave will result in a shift in the RR interval on the ECG (most common cause of a "pause" during NSR), followed by restoration of a normal RR interval
Supraventricular tachycardia's A heart rate of over 100 bpm caused above the ventricle, either the atria, SA node, or AV junction. Includes: Sinus Tach, Atrial tach, Atrial flutter, Atrial fibrillation, and junctional tach.
junctional escape rhythmthe most common cause of this rhythm in healthy individuals is sinus bradycardia.it may also be seen in the presence of a high degree or complete av block.if the ventricular rate is slow, hemodynamic compromise may occur. Regular R-R interval, P waves may be seen before QRS (will be short & inverted), may be buried in the QRS, or may be seen after QRS. treament depends upon the underlying cause and the baseline dysrhythmias . atropine or a pacemaker may be used to increase the ventricular rate
Idioventricular rhythm Rhythm: Regular
Rate: 20-40
P-waves: Absent
PR-Interval: Absent
QRS Complex: Wide
*SA and AV nodes have failed, just have ventricle firing, dying heart, no atrial contraction. Atropine, pacing
Accelerated idioventricular rhythm Same as Idioventricular Rhythm, only faster
-Rate: 40-100
-Rhythm: Regular
-P: None
-QRS: Wide b/c ventricles are only pacemakers. Looks like slow VT
non-sustained ventricular tachycardia lasts for ≤ 30 seconds, rapid ventricular rate
sustained ventricular tachycardia if run of VT lasts longer than 30 sec, can lead to VF
first degree heart block - Appears Sinus
- Exception: P-R interval > 0.20 seconds
second degree heart block, Mobitz type 1 Wenckebach -Progressively longer PR intervals until a QRS is dropped
-Rhythm: Irregular!
-Ventricular Rate: Slow d/t dropped QRS beats
-Tx: Usually benign; if bradycardia and symptomatic, give Atropine
Second degree heart bock, Mobitz Type 2 PR intervals are consistant, but some P waves not followed by QRS complexes, problem in AV node, skipped ventricular beats, can lead to complete heart block
complete heart block a condition in which the AV node is non functional and no impulses from SA node reach the ventricles
multifocal atrial tachycardia Irregular atrial rate greater than 100 bpm with at least 3 morphologically distinct P waves (P's will all have diiferent appearance)

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