Anatomy 2: Exam 1: Part 2
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29 terms
Terms | Definitions |
|---|---|
Pulmonary Valve | Sounds heard in intercostal space at left sternal margin. |
Mitral valve | Sounds heard over heart line with middle of clavicle. |
Tricuspid valve | Sounds heard typically in right sternal margin of 5th intercostal space. |
Blood Flow is controlled entirely by | Pressure changes. |
Blood flows down a | Pressure gradient through any available opening. |
Blood is dependent on | -Properly functioning valves-Conduction system working correctly -Healthy heart muscle tissue. |
Cardiac output (CO) | Heart rate (HR)xStroke volume (SV). |
Preload | Degree to which cardiac muscle cells are stretched just before they contract (critical factor controlling SV, according to Frank Sterling Law of Heart). |
Stroke volume is affected by | Venous return. |
Contractility | Increases SV, lowers ESV. |
Stroke volume is affected by | Increased sympathetic stimulation and chemicals. |
Afterload | Back pressure exerted by arterial blood. |
High blood pressure | Blood backs up, increased ESV and reduced stroke volume. |
Homeostatic Imbalances | -Coronary Atherosclerosis-Hypertension -Multiple myocardial infartions (MI's) -Hypovolemic shock |
Ischemia | Heart muscle is deprived of oxygen. |
Infarction | Death of heart muscle cells |
Fibrillation | Rapid and irregular or out of phase contraction of heart muscle cells. |
Myocarditis | Inflammation of the cardiac muscle layer. |
Ventricular tachycardia | Rapid ventricular contractions that are not coordinated with atrial activity. |
1st age related changes | Sclerosis and thickening of the valve flaps, producing heart murmurs. |
2nd age related changes | Decline of cardiac reserve decline in maximum heart rate. |
3rd age related change | Fibrosis of cardiac muscle, becoming stiffer and impeding nerve conduction. |
4th age related change | Atherosclerosis- Formation of lipid deposits on artery walls. |
Coronary atherosclerosis | Clogged arteries impairs blood and oxygen delivery to the heart, heart becomes hypoxic and contract ineffectively. |
Persistent Hypertension | Aortic diastolic pressure > 90mm Hg causes myocardium to work harder, ESV rises. |
Multiple myocardial infarts | Dead heart cells replaced by noncontractile fibrous (scar) tissue. |
Failure can occur on either side | Right and left. |
Right side heart failure | Backs up in peripheral system producing congestion. |
Left side failure | Backs up in lungs, producing pulmonary congestion, or even pulmonary edema. |
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