Anatomy 2: Exam 1: Part 2

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ninauche  on October 2, 2011

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Anatomy 2: Exam 1: Part 2

Pulmonary Valve
Sounds heard in intercostal space at left sternal margin.
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Pulmonary Valve Sounds heard in intercostal space at left sternal margin.
Mitral valve Sounds heard over heart line with middle of clavicle.
Tricuspid valve Sounds heard typically in right sternal margin of 5th intercostal space.
Blood Flow is controlled entirely by Pressure changes.
Blood flows down a Pressure gradient through any available opening.
Blood is dependent on -Properly functioning valves
-Conduction system working correctly
-Healthy heart muscle tissue.
Cardiac output (CO) Heart rate (HR)xStroke volume (SV).
Preload Degree to which cardiac muscle cells are stretched just before they contract (critical factor controlling SV, according to Frank Sterling Law of Heart).
Stroke volume is affected by Venous return.
Contractility Increases SV, lowers ESV.
Stroke volume is affected by Increased sympathetic stimulation and chemicals.
Afterload Back pressure exerted by arterial blood.
High blood pressure Blood backs up, increased ESV and reduced stroke volume.
Homeostatic Imbalances -Coronary Atherosclerosis
-Hypertension
-Multiple myocardial infartions (MI's)
-Hypovolemic shock
Ischemia Heart muscle is deprived of oxygen.
Infarction Death of heart muscle cells
Fibrillation Rapid and irregular or out of phase contraction of heart muscle cells.
Myocarditis Inflammation of the cardiac muscle layer.
Ventricular tachycardia Rapid ventricular contractions that are not coordinated with atrial activity.
1st age related changes Sclerosis and thickening of the valve flaps, producing heart murmurs.
2nd age related changes Decline of cardiac reserve decline in maximum heart rate.
3rd age related change Fibrosis of cardiac muscle, becoming stiffer and impeding nerve conduction.
4th age related change Atherosclerosis- Formation of lipid deposits on artery walls.
Coronary atherosclerosis Clogged arteries impairs blood and oxygen delivery to the heart, heart becomes hypoxic and contract ineffectively.
Persistent Hypertension Aortic diastolic pressure > 90mm Hg causes myocardium to work harder, ESV rises.
Multiple myocardial infarts Dead heart cells replaced by noncontractile fibrous (scar) tissue.
Failure can occur on either side Right and left.
Right side heart failure Backs up in peripheral system producing congestion.
Left side failure Backs up in lungs, producing pulmonary congestion, or even pulmonary edema.

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