Cardiac Auscultation

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Created by:

MDE_GoPens  on December 5, 2011

Description:

Dr. Colyer's Lecture on 12/5/2011

Classes:

UT COM 2014

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Cardiac Auscultation

Diaphragm
High Frequency sounds
1/37
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Definitions

Diaphragm High Frequency sounds
Bell Low Frequency Sounds
Hand Grip Maneuver & Squatting Increases Left Ventricular Volume
Valsalva Maneuver & Standing Decreases Left Ventricular Volume
S1 Heart Sounds Mitral and Tricuspid Components
S2 Heart Sounds Aortic and Pulmonic Components
Increased Intensity of S1 Tachycardia
High Output States (e.g. fever)
Mitral Stenosis (mitral valve open at onset of ventricular systole with sudden closure)
Decreased Intensity of S1 1st Degree Block (Long PR)
Cardiomyopathy
S1 Splitting Listen At LLSB
Seen in RBBB
S2 Splitting - Physiologic Due to Respiration
Inspiration will decrease intrathoracic pressure leading to increased filling of RV and longer RV systole
PV Closes after AV
S2 Splitting - Paradoxical Delayed onset (LBBB, RV pacing)
Prolongation (Aortic Stenosis)
P2 is before A2
May resolve with inspiration
S2 Splitting - Fixed ASD
VSD
RV Failure
Ejection Click Cause: Sudden upward doming of semi-lunar valve
Listen: Apex
DDx: Aortic stenosis, pulmonic stenosis
Only Right-Sided Sound to Decrease in Intensity upon Inspiration Pulmonic Ejection Click
Mid Systolic Click Late in Systole
Mitral Valve Prolapse
Increase LV (hand grip) = later
Decreased LV (valsalva) = earlier
Metallica Click Prosthetic Heart Valve
Type: probably St. Jude Valve
Opening Snap Mitral Stenosis
Column of blood from LA runs into fused commisures of MV
Heard at Base
Gauging Severity of Mitral Stenosis? Closer to S2 - More Severe
<0.07 - very severe
S3 Gallop - Normal Young people
High Output States (anemia, pregnancy)
S3 Gallop - LV Ventricle resists filling as blood "slaps" into it
Cardiomyopathy
Mitral Regurgitation
Aortic Regurgitation
S3 Gallop - RV RV enlargement/failure
Pulmonary Embolism
Pulmonary HTN
S4 "Pre-Diastolic" Sound
Stiff ventricle (always pathologic)
Causes: LVH, Severe HTN, Aortic Stenosis, Acute Coronary Syndromes
Characterizing a Murmur Timing: Systolic, Diastolic, Continuous
Intensity: I-VI
Shape: (De)Crescendo, Plateau)
Location of Max Intensity
Pitch
Quality
Radiation
Causes of Systolic Murmur Mitral Regurgitation
Aortic Stenosis
Hypertrophic Obstructive Cardiomyopathy
Pulmonic Stenosis
Small Muscular VSD
Mitral Regurgitation - Causes Endocarditis, Ischemia, MVP
Mitral Regurgitation - Characteristics Systolic Murmur
High Frequency
Holosystolic
Intensity does NOT CHANGE with cycle length
Plateau murmur
Loudest at Apex
Radiates around back to Triangle
Intensifies with handgrip (increase LV)
Aortic Stenosis - Causes Congenital
Rheumatic
Degenerative/Calcific
Aortic Stenosis - Characteristics Systolic Murmur
Heard at right base
Crescendo-decrescendo
Intensity varies with cycle length
Loss of A2 - severe
Gallavardin Effect Musical quality of Aortic Stenosis heard at Apex during systole
Diastolic Murmurs Aortic Insufficiency
Mitral Stenosis
Austin-Flint
Graham Steele
Tricuspid Stenosis
Aortic Insufficiency - Causes Congenital, Rheumatic, Endocarditis, VSD, CVD
Aortic Insufficiency - Characteristics High Frequency
Decrescendo
3-4 Left Intercostal Space
Radiation implies Marfans
Often co-morbid with Aortic Stenosis
Mitral Stenosis - Characteristics Low Frequency
Rumble
Heard at Apex
Loud S1
Opening Snap Present (unlike Austin-Flint)
Austin-Flint Murmur Aortic Regurgitation
AR Jet causes turbulence of mitral inflow jet, thus mimicking mitral stenosis
NO opening snap
Graham-Steele Murmur Pulmonary Insufficiency
Heard in Pulmonic Area
Patent Ductus Arteriosis Continuous Murmur
Differential cyanosis
Left Base
Envelops S2
Pericardial Friction Rub Triple Phased
Scratchy/Leathery
Pathology: apposition of abnormal visceral and parietal pericardia
Exacerbated by: push hard with stethoscope, bending forward, held exhalation

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