Cardiac Auscultation
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Created by:
MDE_GoPens on December 5, 2011
Description:
Dr. Colyer's Lecture on 12/5/2011
Classes:
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37 terms
Terms | Definitions |
|---|---|
Diaphragm | High Frequency sounds |
Bell | Low Frequency Sounds |
Hand Grip Maneuver & Squatting | Increases Left Ventricular Volume |
Valsalva Maneuver & Standing | Decreases Left Ventricular Volume |
S1 Heart Sounds | Mitral and Tricuspid Components |
S2 Heart Sounds | Aortic and Pulmonic Components |
Increased Intensity of S1 | TachycardiaHigh Output States (e.g. fever) Mitral Stenosis (mitral valve open at onset of ventricular systole with sudden closure) |
Decreased Intensity of S1 | 1st Degree Block (Long PR)Cardiomyopathy |
S1 Splitting | Listen At LLSBSeen in RBBB |
S2 Splitting - Physiologic | Due to RespirationInspiration will decrease intrathoracic pressure leading to increased filling of RV and longer RV systole PV Closes after AV |
S2 Splitting - Paradoxical | Delayed onset (LBBB, RV pacing)Prolongation (Aortic Stenosis) P2 is before A2 May resolve with inspiration |
S2 Splitting - Fixed | ASDVSD RV Failure |
Ejection Click | Cause: Sudden upward doming of semi-lunar valveListen: Apex DDx: Aortic stenosis, pulmonic stenosis |
Only Right-Sided Sound to Decrease in Intensity upon Inspiration | Pulmonic Ejection Click |
Mid Systolic Click | Late in SystoleMitral Valve Prolapse Increase LV (hand grip) = later Decreased LV (valsalva) = earlier |
Metallica Click | Prosthetic Heart ValveType: probably St. Jude Valve |
Opening Snap | Mitral StenosisColumn of blood from LA runs into fused commisures of MV Heard at Base |
Gauging Severity of Mitral Stenosis? | Closer to S2 - More Severe<0.07 - very severe |
S3 Gallop - Normal | Young people High Output States (anemia, pregnancy) |
S3 Gallop - LV | Ventricle resists filling as blood "slaps" into itCardiomyopathy Mitral Regurgitation Aortic Regurgitation |
S3 Gallop - RV | RV enlargement/failurePulmonary Embolism Pulmonary HTN |
S4 | "Pre-Diastolic" SoundStiff ventricle (always pathologic) Causes: LVH, Severe HTN, Aortic Stenosis, Acute Coronary Syndromes |
Characterizing a Murmur | Timing: Systolic, Diastolic, ContinuousIntensity: I-VI Shape: (De)Crescendo, Plateau) Location of Max Intensity Pitch Quality Radiation |
Causes of Systolic Murmur | Mitral RegurgitationAortic Stenosis Hypertrophic Obstructive Cardiomyopathy Pulmonic Stenosis Small Muscular VSD |
Mitral Regurgitation - Causes | Endocarditis, Ischemia, MVP |
Mitral Regurgitation - Characteristics | Systolic MurmurHigh Frequency Holosystolic Intensity does NOT CHANGE with cycle length Plateau murmur Loudest at Apex Radiates around back to Triangle Intensifies with handgrip (increase LV) |
Aortic Stenosis - Causes | CongenitalRheumatic Degenerative/Calcific |
Aortic Stenosis - Characteristics | Systolic MurmurHeard at right base Crescendo-decrescendo Intensity varies with cycle length Loss of A2 - severe |
Gallavardin Effect | Musical quality of Aortic Stenosis heard at Apex during systole |
Diastolic Murmurs | Aortic InsufficiencyMitral Stenosis Austin-Flint Graham Steele Tricuspid Stenosis |
Aortic Insufficiency - Causes | Congenital, Rheumatic, Endocarditis, VSD, CVD |
Aortic Insufficiency - Characteristics | High FrequencyDecrescendo 3-4 Left Intercostal Space Radiation implies Marfans Often co-morbid with Aortic Stenosis |
Mitral Stenosis - Characteristics | Low Frequency Rumble Heard at Apex Loud S1 Opening Snap Present (unlike Austin-Flint) |
Austin-Flint Murmur | Aortic RegurgitationAR Jet causes turbulence of mitral inflow jet, thus mimicking mitral stenosis NO opening snap |
Graham-Steele Murmur | Pulmonary InsufficiencyHeard in Pulmonic Area |
Patent Ductus Arteriosis | Continuous Murmur Differential cyanosis Left Base Envelops S2 |
Pericardial Friction Rub | Triple PhasedScratchy/Leathery Pathology: apposition of abnormal visceral and parietal pericardia Exacerbated by: push hard with stethoscope, bending forward, held exhalation |
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