Rehab II: Cerebellar and Basal Ganglia Disorders

About this set

Created by:

skyliele  on April 29, 2009

Subjects:

rehabilitation, rehab 2, Physical Therapy

Classes:

Physical Therapy Study

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Rehab II: Cerebellar and Basal Ganglia Disorders

Two parts of the forebrain
Telencephalon, Diencephalon
1/48
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Two parts of the forebrain Telencephalon, Diencephalon
2 divisions of the peripheral nervous system autonomic and somatic
Diencephalon includes Thalamus, Hypothalamus
Cerebellum operates in a feed___ direction feed-forward
4 processes of recovery after damage to nervous system Plasticity, Sparing, Compensation (substitution), Recovery of tissue
Substitution after damage to the nervous system is using what you have left to do the skill
Sparing is training what is left in the nervous system
Cerebellum compares intended activity with the actual changes occurring in the body and then initiates corrective adjustments
3 parts of the cerebellum Spino, Cerebro, Vestibulocerellum
Vestibulocerebellum receives input from the vestibular receptors as well as vestibular nuclei
Vestibulocerebellum coordinates head movement with eye movement
Spinocerebellum regulates posture and controls axial movments as well as ongoing execution of limb movements. Compensates for small variations in load
Spinocerebellum receives somatosensory input from the Spinal cord and trigeminal nuclei
Cerebrocerebellum gets extensive input from sensory and premotor cortices via pontine nuclei.
Cerebrocerebellum is important for __ movements multi-joint
planning, timing, learning new skilled movement occurs in what part of the cerebellum cerebrocerebellum
Dysmetria, ataxia and hypotonia are signs of Cerebellar lesion
Hypotonia is low muscle tome
Adiadokokinesia is the complete inability to coordinate antagonist
Clinical signs of cerebellar dysfunction hypotonia, intention tremor, poor balance and gait, nystagmus, dysdiadokokinesia, dysmetria, ataxia
The basal ganglia includes these three parts the striatum, the substantia nigra and the subthalamic nucleus
The Striatum includes the caudate nucleus, globus pallidus, putamen
The Basal ganglia does not control movement directly. It does not have a motor pathway to the spinal cord of its own.
The basal ganglia controls movement via the thalamus, cerebral cortex, cerebellum and brainstem
Basal ganglia function in the planning, sequencing and initiation of movement
Lesions to the basal ganglia may cause rigidity, tremor, ballism, dystonia, chorea
Dystonia is prolonged muscle contractions with difficulty relaxing the contraction
chorea is rapid irregular movements
2 diseases of the basal ganglia Parkinson's and Huntington's
Huntington's disease is characterized by rapid choreiform jerky involuntary movements, dementia and is hereditary dominant
50% of offspring inherit Huntington's disease
Huntington's disease usually starts with psychological symptoms including depression, memory impairment, anxiety and sexual probs
Parkinson's is a slow progressive disease caused by a depletion of dopamine, which is produced by the substantia nigra
4 non-idiopathic etiologies of Parkinson's: Infectious (encephalitis lethargica); toxins (industrial poisons, synthetic heroin); pharmacologic (drugs producing Parkinson's like symptoms); degenerative diseases of CNS
Patients with Parkinson's have bradykinesia which is movement reduced in speed, range, and amplitude. Can also mean difficulty initiating movement
Leadpipe rigidity Smooth constant resistance to passive movement
Resting tremors stop with movement
Decreased postural reactions can be caused by Parkinson's
Hypomimia is mask like face
Festinating gait is increase in walking speed due to forward leaning trunk and displaced center of gravity.
Propulsive gait is... forward acceleration
retropulsive gait is... backward acceleration
micrographia is very small hard to read handwriting
Dementia occurs in this % of Parkinson's patients 35 to 40%
Dysphagia, ANS dysfunction and cogwheel rigidity are signs of Parkinson's disease (three things)
Deep brain stimulation is a Parkinson's treatment for this type of patient one whose meds don't work as well as before or not enough; DBS is better for younger people than older
Brain implants work via 4 electrodes in the thalamus and they block malfunctioning brain signals (tremors). Can be turned on/off with a magnet
Good PT options for Parkinson's Gait training, rotation, gentle rocking, PNF, AROM and PROM, breathing exercises

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