Spring Anatomy Heart

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rachy1191  on January 29, 2012

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Spring Anatomy Heart

heart
continually pumps blood with oxygen, nutrients, and wastes to all cells of the body
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heart continually pumps blood with oxygen, nutrients, and wastes to all cells of the body
pericardium double-walled sac enclosing the heart, protects the heart, anchors it, and prevents blood from over filling the heart
outer- (parietal) fibrous made of dense connective tissue
inner- (visceral) serous, epicardium
pericardial cavity between the parietal and visceral layer of the serous pericardium, contains a film of fluid that allows the membranes to slide smoothly during heart activity
pericaritis inflammation of the pericardium that makes the serous membrane rough and creates a creaking sound when listened to in a stethoscope (pericardial friction rub)
epicardium visceral layer of the serous pericardium, simple squamous with areolar connective beneath
myocardium bulk of the heart that contracts (cardiac muscle), each muscle is connected by branches of connective tissue called Fibrous Skeleton of the Heart, reinforces and anchors
endocardium lines the heart chambers and covers valves, continuous with the epithelial linings of blood vessels entering and leaving the heart
atria chambers of the heart, a left and a right, receiving chambers
right atrium shallow depression called the fossa ovalis, blood enters from the superior vena cava, inferior vena cava, and coronary sinus
left atrium blood enters from the pulmonary veins
auricles protruding appendages that increase atrial volume
pectinate muscles bundles of muscles on the inferior portion of the atria
ventricles chambers of the heart, a left and a right, discharging chambers, internal walls have irregular ridges of muscles called trabeculae carnae, papillary muscles connect to valves by chordae tendinae
right ventricle blood leaves through the pulmonary trunk (artery) of the lungs
left ventricle blood leaves through the aorta to the body
tricuspid valve btwn the right atrium and right ventricle
bicuspid valve mitral, btwn the left atrium and left ventricle
coronary circulation keeping the heart supplied with blood
coronary arteries peak blood flow occurs when ventricles relax, blood in the aorta briefly surges toward the heart filling the coronary arteries
angina pectoris deficiency of blood delivery to the heart, stress induced spasms of the coronary arteries cause pain, myocardial cells are weakened
myocardial infarction heart attack, prolonged coronary blockage, myocardial cells die, most area of cell death are repaired with scar tissue
heart valves flow through the heart is unidirectional because of heart valves
atroventricular vavles (AV valves), R- tricuspid valve, L- bicuspid valve, collagen chords attach to the valve flaps to papillary muscles
semilunar valves pulmonary semilunar valve and aortic semilunar valve
cardiac cells striates, short, branched and interconnected, 1 or 2 nuclei, less SR than skeletal muscle, larger T-tubules than skeletal, lots of mitochondria
intercalated disc connect the plasma membranes of adjacent cardiac cells, made of desmosomes and gap junctions
desmosomes hold cells together, prevent cardiac cells from pulling apart during contraction
gap junctions form channels that allow flow of ions from the sarcoplasm of one cell to a connecting cell allowing electrical stimulation
fascia adherins thick band of myofilaments, anchor to plasma membrane
cardiac contraction more mitochondria means it depends greatly on oxygen for metabolism, relies on aerobic respiration, uses glucose and fatty acids as fuel molecules but can switch to whatever is available for use, not prone to fatigue
autorhythmic cells do not maintain stable resting potential, continually depolarize-Pacemaker Potentials
Pacemaker Potential initiate the action potentials that spread throughout the heart to trigger contraction
Sinoatrial Node (SA Node), right atrium wall, fastest depolarization rate in the myocardium, pacemaker of the heart
Atroventricular Node (AV Node) located in the interatrial septum, depolarization wave spreads throughout the atria and via the internodal pathway
Bundle of His Atriventricular Bundle, interventricular septum, R and L atrioventricular bundle branches, goes through the interventricular septum toward the apex
Purkinje Fibers specialized fibers that run through the ventricular walls, directly supplies the papillary muscle, contracts before the ventricles contract
arrhythmias and fibrilation rapid and irregular contractions; control of heart rhythm is take away from the SA node
ectopic focus any region other than SA node that fires
Autonomic Nervous System modify heart rate
sympathetic- adrenergic, increase rate and force of heart beat
parasympathetic- cholinergic, slows heart rate
epinepherine released by adrenal medulla, produces same cariac effects as norepinepherine
thyroxine released from thyroid, increases metabolic rate and body heat production, causes a slower more sustained increase in heart rate than epinepherine and enhances the effects of epinepherine and norepinepherine
ions plasma electrolytic imbalances pose danger to the heart
Ca++ imbalance effects heart rate
K+ imbalance can be life threatening
Electrocariograph ECG, detects electrical current generated and transmitted through the heart
electrocardiogram record of the electrical current through the heart, 'picture' of the heart's activity
heart sounds lub- dub, closing of valves
lub- AV closes
dub- semilunar closes
heart murmur abnormal heart sounds, common in children bc heart walls are still relatively thin and vibrate more easily when the blood moves, indicate valve trouble
tachycardia abnormally FAST heart beat, greater than 100 beats per min, promotes fibrillation
results from: elevate temp, stress, drugs, disease
bradycardia heart beat is too LOW, lower than 60 beats per min, desirable consequence of endurance training- resting rate may be lower but the heart is still putting out the same output
in poorly conditioned people- can lead to bad blood circulation
cardiac output CO, amount of blood ejected from each ventricle in one min, not constant, varies with activity
coronary atherosclerosis heart failure- blockage of coronary vessels with fatty build up, cant deliver nutrients/waste, contractions are less effective
persistent high blood pressure heart failure- aortic diastole, L ventricle exerts only slightly over the amount its supposed to, has to work a lot harder to open valves and pump
multiple myocardium infarct heart failure- pumping defficiency is depressed bc the heart cells are replaced by scar tissues
dilated cardiomyopathy DCM, heart failure- ventricles stretch and become flabby, myocardium deteriorates, heart working too hard and not getting good output
pulmonary congestion heart failure- L side of heart fails so R side sends blood to the lungs but L side is no longer circulating, so blood is stuck in lungs, pressure increases and fluid leaks into the lungs, suffocation
peripheral congestion heart failure- blood stagnates in organs, impairs cells to obtain nutrients/oxygen, noticeable in feet, fingers, ankles

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