| Term | Definition |
|
sinosodial |
is the pattern that consist of a series of cycles that are extremely smooth and regular in aptitude and duration |
|
sinosodial |
these patterns are associated with sever hypoxia, RH isoimmunization, severeanemia, abruptio placentae, Fetal-maternal hemorrhage, severe feta acidosis, and chronic fetal bleeding |
|
Variability |
flunctuations or change in heart ragte; reflects interplay of PNS and SNS |
|
Rhythmic |
are the type of fluctuations that are measured over 1 min and occurs 2-6 times per min with a normal rage of 6-10bpm; |
|
Variability |
disappears when there is fetal O2 reserve, and disappears if there is fetal O2 deficit to CNS |
|
absent |
is the type of variability that no amplitude detected 0 bpm |
|
minimal |
is the type of variability where amplitude deteced but 5 bpm |
|
moderate |
is the type of variability that is 6-25 |
|
marked |
is the type of varability of >25 bpm |
|
saltatory |
is the pattern that has marked or excessive variability, and if it is in the absence of other fetal concerns this pattern is not thought to be indicated fetal distress |
|
acceleration |
periodic increased in baseline FHR |
|
decelleration |
periodic decrease in baseline FHR |
|
tachycardia |
baseline FHR is greater than 160 BPM continuing for 10 min or more |
|
bradycardia |
baseline FHR less than 120 bpm continuing for 10 min or more |
|
Long Term Variability |
measured over 1 min; occurs 2-6 times per min with a normal range of 6-10 bpm; increased by fetal movement; decreased or absent with fetal sleep or hypoxia |
|
short term variability |
indicated CNS functionn, disappears if there is fetal O2 deficit to CNS; indicator of fetal O2 reserve, measured only by inernal fetal monitoring |
|
periodic |
these accelerations usually accompany contractions |
|
spontaneous |
these types of accelerations are symmetric, uniform and nonperiodic; represent inctact CNS, not associated with contractions or decelleration |
|
early |
type of decelleration that the onset begins with onset of contractions, return to baseline by end of contraction, uniform shape, upsidedown contraction |
|
early decelleration |
is caused by head compression, increased intracrainal pressure or due to pressure on fetal head as it progresses down birth canal |
|
late decelleration |
late in onset and recovery, Begin at or within few seconds after peak of contraction, return to baseline well after contraction is over. |
|
late decelleration |
is caused by utero-placental insufficency, maternal hypotension, uterine hypertonus, and placental pathology |
|
late decelleration |
is managed by turning to left side, increase IV fluid rate, O2, discontinue pitocin, notify dr., |
|
variable decelleration |
vary in onset, occurance and appearance from contraction to contraction, pattern may be 'V' r wide square shaped. |
|
variable decelleration |
could be caused by cord compression, frequently seen in nuchal cord, short cord, or prolapsed cord |
|
variable decelleration |
managed by relieve pressure on cord, change naternal position, |
|
prolapsed decellerations |
FHR decliens form baseline for 2-10 min; may occur suddenly |