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5 Written questions

5 Matching questions

  1. Serotonin receptor agonist MOA
  2. alpha 2-adrenergic agonist DDI
  3. Serotonin receptor agonist
  4. anti-histamines AE
  5. alpha 2-adrenergic agonist EE
  1. a Causes CNS depression with opiates, sediatives, hypnotics, anesthetics, alcohol.
  2. b binds the neuroreceptors for serotonin and dopamine in the brain and increases norepinephrine metabolism to relieve anxiety.
  3. c Buspirone (Buspar)
  4. d RARE
  5. e decrease anxiety

5 Multiple choice questions

  1. decrease anxiety-induced physiology response, such as tachy and hypertension.
  2. do not take with alcohol
  3. Not for patient having lung problems (asthma, COPD, etc), heart block, bradycardia, diabetes mellitus
  4. Do not discontinue abruptly,
  5. Clonidine (Catapres)

5 True/False questions

  1. alpha 2-adrenergic agonist AECHF, hallucinations, delirium


  2. Non-benzodiazepine Sedative-hypnotic DDIadditive effect with CNS depressants (e.g., sedatives, psychotropic drugs, anticonvulsants, antihistamines, alcohol)


  3. Benzo-diazapines GEROpotentiate the action of gamma-aminobutyric or GABA, including any other inhibitory transmitters in the CNS resulting in decreased anxiety. Most agents cause a decrease in CNS excitability


  4. Beta blocker USEUse for patients facing anxiety-producing events such as performing on stage, making a speech, and test anxiety. Allows for cognition unlike Benzodiazepines


  5. Non-benzodiazepine Sedative-hypnotic AE• depression, worsening
    • suicidal ideation
    • aggressive behavior
    • complex sleep-related behavior
    • hallucinations
    • amnesia
    withdrawal if abrupt D/C