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5 Written questions

5 Matching questions

  1. catecholamines
  2. exocytosis
  3. spinothalamic tract
  4. oligodendrocytes
  5. iontrophic receptor
  1. a - in CNS
    - provide myelin sheaths around axons in the brain & spinal cord
  2. b - DRGN enters dorsal horn at each spinal cord level
    - pain, temp, some touch
    - ascend/descend in lissaur's tract
    - synapse in substanita gelatinosa
    - decussate in ventrcal spinal cord
    - ascend ventrally in spin cord as spinothalamic tract
    - synapse in thalamus
    - thalamic axons travel to Si somasensory cortex and synapse in layer 4
  3. c - with thier associated proteins form one complex
  4. d - a calcium dependent process
    - membrane bound individual vesicles fuse with the plasma membrane
    - spill the intracellular contents of the vesicle into the extracellular space, referred to as the synaptic cleft
  5. e - DA, NE, Epi
    - tryosine -> l-dopine - > DA -> NE -> Epi
    - degraded by monosamineoxidase MAD
    - DA: pleasure seeking behavior; addiction; cocain, amphatamine
    - parkinson's disease: lack of DA in substrate nigria, treated w/l-dops; DA cannot cross bbb

5 Multiple choice questions

  1. - exocytosis
    1. presynaptic ap
    2. deoplarization of synamtic terminal
    - released by the first neuron at the presynaptic terminal, release site: active zone
    - triggered by the arrival of an ap in the axon terminal
    - released nt is bounded by the second neuron at the postsynaptic membrane
    - leads to transient depolarizaton of the membrane
  2. - another name for an association neuron
  3. - processes sensory information from muscle and skin
  4. - follows an ap
    - cannot be fired even if there is a transient depolarization
    - due to Na channel inactivation, h gate slow to reopen
    - membrane is negative for h gate to reopen
    - ends when the K channel closes
    - subquent ap cannot be generated until membrane is repolarized
  5. - IPSPs
    - precursors serine (prefered) glucose (for all IPSP)
    - less common inhibitory NT
    - stychine: antagonist (blocks CL- channels) to gly receptor on Cl- channel
    -> effects: uncontrolled depolorization in the brain (no inhibition)

5 True/False questions

  1. scale tympani- bounded by basilar & ressner's membranes
    - has endolymph fluid


  2. amino acids- start with "g"
    - also aspartate
    - made in axon terminus (synaptic vesicles


  3. otto lowei- chemical nature of nerve transmission
    - proved that some soluble chemical released by the vagus nerve was controlling the heart rate


  4. threshold depolarization- a less negative membrane pontential triggers the firing of an ap


  5. GABA- precursor: acetyl CoA, chloine
    - made by cholineacetyltransferase ChAT
    - degraded by acetylchlolinsterase AChE
    - nicotinic receptor (NAChR) - muscle contraction
    - mesacrarisic receptor - slow down heart rate


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