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7 Written questions

6 Multiple choice questions

  1. -Aspirin/ibuprofin decreases synthesis of prostaglandins
    preventing prostaglandins from sensitizing nociceptors.
  2. - (Sypatheric Reflex Dystrophy) abnormal response to trauma leading to limb disuse; disuse of limb precipitates an increase in levels of neurochemicals that cause peripheral inflammation, along with a decrease in sympathetic regulation of blood flow &swating; symptoms include pain, vascula changes, atrophy affecting entire hand/foot, red/pale skin, excessive sweating, edema, late stage dry/cold skin, muscle atrophy and osteoporosis.
  3. -If peripheral sensory information is completely absent, CNS neurons in the nociceptive pathway become abnormally active
  4. -post amputation, absence of sensory info causes neurons in central nociceptive pathways to become overactive; maladaptive structural reorganization is found in the SC,
    thalamus, and cerebral cortex; there is overlap of cortical reprsentation that are normally separate (loss of sensory info lighting up pain matrix)
  5. -decrease production of neurotransmitters that transmit pain
  6. -occurs in demyelinated areas - GB Syndrome

6 True/False questions

  1. Ectopic Foci-develop at nerve stump end, and tapping induces pain (Tinel's sign); tapping test at end of nerve creates a lot of pain

          

  2. Rhizotomy-severing of the spinothalamic tract

          

  3. Mononeuropathy-involve more than one nerve, such as diabetic neuropathy and Guillian Barre

          

  4. 1) PNS-If peripheral sensory information is completely absent, CNS neurons in the nociceptive pathway become abnormally active

          

  5. Massage-If peripheral sensory information is completely absent, CNS neurons in the nociceptive pathway become abnormally active

          

  6. 3) The Pain Matrix-develop at nerve stump end, and tapping induces pain (Tinel's sign); tapping test at end of nerve creates a lot of pain