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5 Written questions

5 Matching questions

  1. Secretin family
  2. High leptin levels from large fat cells due to eating too much
  3. Neurohypophysial contractile activity
  4. Hyperlipidemia
  5. Neurovascular hypothesis
  1. a gluconeogenesis leads to inc FFA and glycerol; dec VLDK clearance due to dec lipoprotein lipase activity (requires insulin)
  2. b Secretin, glucagon, gastric inhibitory peptide (GIP), vasoactive intestinal peptide (VIP); share AA sequences; inc cAMP
  3. c pregnancy (OT release causes contraction during labor); mammary glands (OT causes milk release); mating (inc uterine activity, orgasm, milk ejection, inc sperm transport)
  4. d hormonal rather than direct neural control of AP gland; Geoffrey Harris supporter;
  5. e stimulate a-MSH release which binds to melanocortin-4 receptor, decreases appetitie/eating

5 Multiple choice questions

  1. insulin release in response to glucose; inhibits gastric acid secretion; anabolic action on adipose cells (FFA to triglycerides);
  2. emaciation from muscle breakdown; use of gluconeogenic AAs for energy
  3. yellow/orange/red pigments; carotenoids/pteridines; located in dermis
  4. roof of mouth -- turns into adeno
  5. increase amount of hormone released: Prolactin (synthesis and release inhibited by dopamine), aMSH (controlled by dopamine)

5 True/False questions

  1. Electrolyte depletionInc K+ in urine

          

  2. Alpha melanocyte stimulating hormone (alpha MSH)in pars intermedia

          

  3. GIP - gastric inhibitory peptidedopamine inhibits MSH release, dopamine present in hypo portal system

          

  4. Fxs of 1,25(OH)2D3insulin release in response to glucose; inhibits gastric acid secretion; anabolic action on adipose cells (FFA to triglycerides);

          

  5. Ghrelinprotein released from stomach; levels rise before meals; experiment with rodents showed that ones treated wtih horm ate more and gained weight; humans treated at 30% more at a buffet

          

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