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5 Written questions

5 Matching questions

  1. Somatomedin hypothesis
  2. Type 2 DM
  3. Polyuria
  4. GH secretion
  5. GIP - gastric inhibitory peptide
  1. a excess urine; glucose acts as diuretic; Na slats of ketone bodies also diuretics; leased to hemoconcentration, circulation failure, oliguria
  2. b Maturity onset, non insulin; most common; insulin level may be low nl or high; insulin normally not required to tx but may facilitate management
  3. c says GH actions are mediated by somatomedins produced in liver and other tissue; Ex: insulin like growth factors (IGF)
  4. d aka glucose-dependent insulinotropic peptide, enterogastrone; made in K cells, small intestine; stimulus for release are fats and glucose
  5. e highest during sleep, hypothalamic control, hypoglycemia stimulus released

5 Multiple choice questions

  1. affects pigmentation via cation of malanophores
  2. emaciation from muscle breakdown; use of gluconeogenic AAs for energy
  3. enzye, acid/base secretion; smooth muscle contraction; horm release from pancreatic islets; growth promoting actions
  4. dec bone density due to demineralization; postmenopausal (type 1) -- dec steroid hormone; senile (type 2) - lack of Vit D :: spinal osteoporosis - loss of mineralization, compression of vertebrae
  5. inc glucose uptake via GLUT-4, glucose storage in glycogen, AA uptake and protein sythesis

5 True/False questions

  1. Large fat cellsproduce more leptin to eat less


  2. Alpha melanocyte stimulating hormone (alpha MSH)hypoglycemia; AA (arginine and alanine blocked by glucose); Epi, norepi


  3. Visceral fat more importantInc K+ in urine


  4. Leptin (greek for thin)released from adipose cells; gene termed -ob for obese


  5. Category 1 hormones (GH, PRL)similar size (191/199); common AA