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5 Written questions

5 Matching questions

  1. pituitary gland aka
  2. Diencephalon
  3. Somatomedin hypothesis
  4. Effects of glucagon on liver
  5. Hyposecretion
  1. a panhypopituitarism - total loss of secretion
  2. b says GH actions are mediated by somatomedins produced in liver and other tissue; Ex: insulin like growth factors (IGF)
  3. c Downgrowth from neurohypophysis
  4. d hypophysis
  5. e inc in glucose release, gluconeogenesis, glycogenolysis, ketogenesis:: dec in glycogenesis

5 Multiple choice questions

  1. induce insulin resistance: Resistin (proinflammatory, TZD's) IL-6 (proinflammatory, inc with obesity and DM) TNF-a (proinflammatory cytokine,r elease by visceral fat more than subcutaneous)
  2. Show little staining (ACTH)
  3. inc breakdown of tri,di,monoglycerides forming FFA and glycerol; simulates hormone dependent lipases
  4. aka glucose-dependent insulinotropic peptide, enterogastrone; made in K cells, small intestine; stimulus for release are fats and glucose
  5. stimulate growth, extracellular deposition of bones, lipases that convert triglycerides to FFAs and body fat

5 True/False questions

  1. Hyperglycemiagluconeogenesis leads to inc FFA and glycerol; dec VLDK clearance due to dec lipoprotein lipase activity (requires insulin)


  2. Visceral fat more importantmore highly associated with metabolic syndrome, releases adipokines: hormones that regulate energy and glucose homeostasis


  3. Location of GI hormsinc tubular reabsorption of Ca into ascending loop of Henle; inc excretion of PO4, inhibits H+ secretion into tubules; inhibits bicarb reabsorption


  4. Color change with backgroundproduce more leptin to eat less


  5. HyperlipidemiaNeoplastic tumor of hormone producing cells, most common in microadenonas