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31 terms

Ch. 10: Muscular Tissue

STUDY
PLAY
regulatory/contractile/structural
three kinds of muscle proteins
regulatory
proteins that switch the contraction process on and off
contractile
proteins that generate ofrce during contraction
myosin
functions as a motor protein which can achieve motion
myosin
convert ATP to energy of motion
myosin
thick filaments
actin
thin filaments
actin
provide a site where a myosin head can attach
structural
proteins that align the thick and thin filaments properly
titan/dystrophin
two types of structural proteins
titan
stabalize the position of myosin
dystrophin
links thin filaments to the sarcolemma
ATP hydrolysis/formation of cross-bridges/power stroke/detachment of myosin from actin
4 steps of the contraction cycle
ATP hydrolysis
step that reorients and energizes the myosin head
formation of cross-bridges
step of the contraction cycle where the myosin head attaches to the myosin-binding site on actin
power stroke
step where the crossbridges rotates, sliding the filaments
detachment of myosin from actin
step where the myosin head detaches from actin
action potentials
causes Ca++ to be released from the SR into the muscle cell
Ca++
moves tropomyosin away from the myosin-binding sites on actin allowing cross-bridges to form
Ca++ pumps
return Ca++ back to the SR quickly
length of sarcomeres
the forcefulness of muscle contraction depends on this
neuromuscular junction/synapse/synaptic cleft/neurotransmitter/synaptic vesicles/motor end plate
structures involved in the contraction process
neuromuscular junction
action potentials arise at the interface of the motor neuron and muscle fiber
synapse
where communication occurs between a somatic motor neuron and a muscle fiber
synaptic cleft
gap that seperates two cells
neurotransmitter
chemical released by the initial cell communicating with the second cell
synaptic vesicles
sacs suspended within the synaptic end bulb containing molecules of the neurotransmitter acetylcholine
motor end plate
the region of the muscle cell membrane opposite the synaptic end bulbs
release of ACh/activation of ACh receptors/production of muscle action potential/termination of ACh activity
nerve impulses elicit a muscle action potential in the following way
strabismus
crossed eyes
botulinum
blocks release of ACh from synaptic vesicles