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5 Written questions

5 Matching questions

  1. Hypnozoite stage is present in
  2. *Clinical symptoms of plasmodium infection
  3. Found in Asia, South America, North Africa
  4. Antigenic variation in plasmodium
  5. Discuss P. ovale
  1. a 1. Severe anemia 2. Cerebral malaria (coma, seizures, deadly in 25-50%) because RBC's become sticky in brain. Cytokines and NO production 3. Placental malaria (infection of the placenta, not transplacental, reduces blood flow to baby - low birth weight and still birth. 4. Can also lead to hypoglycemia and hepatosplenomegally, and pulmonary edema and kidney failure.
  2. b P. vivax
  3. c Mild tertian malaria - 48 hr recurring fevers. Rare. Difficult to diagnose because of similarity to P. vivax. Can relapse - hypnozoite Sch├╝ffner's dots.
  4. d ~50 var genes, only one expressed at a time. var switching can lead to recrudescence.
  5. e P. vivax and P. ovale

5 Multiple choice questions

  1. One merozoite could produce billions of parasites in 2 weeks. Humans can develop tolerance, premunition and keeping parasite at low levels. Infants protected by maternal antibodies.
  2. Found in plasmodium apicomplexans. A pigment they produce as a result of hemoglobin breakdown.
  3. 1. Mosquito injects SPOROZOITE into blood 2. sporozoite invades liver cells and becomes TROPHOZOITE 3. Replication by exoerythrocytic schizogony, trophozoites to MEROZOITES 4. merozoites released and attack RBCs 5. merozoites to RING STAGE TROPHOZOITES (cycles back to merozoites or macrogametocyte) 6. Macrogametocytes and microgametocytes form zygote.
  4. asexual schizogony
  5. heteroxenous - mosquito and hman.

5 True/False questions

  1. You catch this disease more easily during wartime because of breaks in sanitation4 widely known, 1 new recent one.

          

  2. Where do you find plasmodium?Mosquito bite (sporozoite injection) or blood transfusion

          

  3. Two major factors of plasmodium pathogenesis1. Anemia arising from RBC destruction (merozoites released) 2. Host inflammatory response (overproduction of cytokines, fevers and chills)

          

  4. Explain why different types of plasmodium cause different types of fevers?1. Mosquito injects SPOROZOITE into blood 2. sporozoite invades liver cells and becomes TROPHOZOITE 3. Replication by exoerythrocytic schizogony, trophozoites to MEROZOITES 4. merozoites released and attack RBCs 5. merozoites to RING STAGE TROPHOZOITES (cycles back to merozoites or macrogametocyte) 6. Macrogametocytes and microgametocytes form zygote.

          

  5. Why is Plasmodium not as prevalent in temperate regions and developed countries?Easier to control vector in temperature regions and in developed countries there is more money and more advanced infrastructure.