Impregnated females burrow stratum corneum of epidermis to deposit eggs and feces. Causes inflammatory response. 1st infestation 30-60 days after contact. Later infestations, inflammatory response starts in 48 hours. Highly contagious. Rash with various types of lesions (papules, burrows, pustules). Lesions usually in webs of fingers, intergluteal folds, axillae, palms, wrists, inner thighs and waist. Pruritis - secondary infection.
Minute grayish, threadlike lesions (mite burrows, black dot at end of burrow - mite)
Infants - can look like eczema
Transmission by direct contact. Primary complaint is pruritus.
-Chronic, pruritic, papulosquamous skin condition
-T-cell mediated autoimmune disease
-Triggers - infection, stress, skin injury
-Scalp, elbows, knees, umbilicus, genitals
-Chronic, relapsing pruritic. T-cell mediated autoimmune disease. Usually a family history. Epidermis thickens and plaques form with a silvery, scaly appearance. Scalp, elbows, knees, umbilicus, genitals. Triggers include infection, skin injury, stress. Topical corticosteroids and topical vit D. Ultraviolet B phototherapy for children who do not respond to topical tx. Severe - systemic tx with methotrexate, oral retinoids, and cyclosporine.
-High risk for collapse if: found in enclosed space, child unconscious, and facial/thoracic burns.
-Inhalation injury causes: Aspiration, Bacterial pneumonia, Pulmonary edema and insufficiency (fluid overload or ARDS), Emboli
-Usually upper airway initially because: Breathe in hot air, cooled in upper airway,
Reflexive closure of cords with laryngospasm = rarely problems below cords, CO inhalation/toxic substance
Mucosal erythema, edema, sloughing of mucosa, mucopurulent membrane formed, hard to ventilate