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5 Written questions

5 Matching questions

  1. Anemic infarct
  2. 4 Arterial Thrombi outcome
  3. Arterial Thrombi Morphology
  4. Acidosis
  5. Infarction
  1. a white; organs with single blood supply
  2. b 1. resolution
    2. organization/incorporation
    3. embolization (arterial)
    4. propagation
    *all similar to venous thrombi
  3. c ischemic necrosis of tissue distal to an area of arterial occulsion or in an area of obstructed venous outflow
  4. d adherent masses of blood that have areas of pale alternating with areas of red - lines of zahn
  5. e mixed acidosis occurs. result of renal, anaerobic glycolosis, and respiratory insufficiency.

5 Multiple choice questions

  1. increased inflow into a vessel; i.e. exercise, inflammation
  2. a sudden onset of fibrin thrombi in the microcirculation with consumption of coagulation factors and formation of fibrin degradation products. Potential complication with widespread of thrombin.
  3. -early of compensated shock
    -decompensated, but reversible shock
    -irreversible shock
  4. Allergen (bee sting), neurogenic (spinal cord, pain from trauma), and bacterial endotoxins (septic shock)
  5. when early shock fails: hypotension occurs, BP and CO fall. Tachypnea and SOB lead to heart failure and pulmonary edema causing anoxia, lead to ARDS.

5 True/False questions

  1. Pathology of shock(upon autopsy) interal organs are congested and wet from edema; the lungs are 2-3 times heavier. the liver is congested and blood oozes from it. the intestines are dark from blood pooling and wet. the kidneys are swollen, pale, and congested. and the brain is edematous, flattened gyri.


  2. Sequelaesudeen death, clinically silent; resolution, organization, dyspnea, pulmonary infarct


  3. Edemahypoperfusion of tissues; the circulatory system can no longer supply nutrients and oxygen to peripheral tissues.


  4. Shockhypoperfusion of tissues; the circulatory system can no longer supply nutrients and oxygen to peripheral tissues.


  5. Cardiogenic shockpump failure; i.e. secondary to MI, conduction block or arrythmia, myocarditis or valvular heart disease


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