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5 Written questions

5 Multiple choice questions

  1. consumption of large quantities of alcohol after fasting or being malnourished-->risk of becoming hypoglycaemic

    large quantities of alcohol produce large amount of NADH-->favor lactate formation rather than gluconeogenesis
  2. result of effects of epinephrine (palpitations, tremors, sweating), usually occur following sudden drop in blood glucose levels--catecholamine surge
  3. for diagnosis of hypoglycemia
    1. adrenergic /neuroglycopenic symptoms
    2. blood glucose <50mg/dL at time of symptoms
    3. improvement of symptoms on ingestion of glucose
  4. tumor in region of β-cells of islets of Langerhans, associated with excessive insulin production and secretion, mostly benign solitary lesions,usually found in tail of pancreas, treatment by surgery wherever possible
  5. indicative of insulinoma

5 True/False questions

  1. factitious hypoglycaemiausually occurs 6-7 hours after last meal, indicative of underlying disorder, most patients are Type 1 diabetics receiving too large dose of insulin, may be caused by liver disease (more than 80% of liver damage can severely compromise hepatic gluconeogenesis), can be caused by Addison's disease--hyposecretion of cortisol rendering tissues more sensitive to insulin

          

  2. fasting hypoglycemiausually occurs 6-7 hours after last meal, indicative of underlying disorder, most patients are Type 1 diabetics receiving too large dose of insulin, may be caused by liver disease (more than 80% of liver damage can severely compromise hepatic gluconeogenesis), can be caused by Addison's disease--hyposecretion of cortisol rendering tissues more sensitive to insulin

          

  3. glucagonomatumor in region of β-cells of islets of Langerhans, associated with excessive insulin production and secretion, mostly benign solitary lesions,usually found in tail of pancreas, treatment by surgery wherever possible

          

  4. neuroglycopenic symptoms of hypoglycemiamediated by impaired supply of glucose to brain over extended period of time, following gradual decline in blood glucose concentration, does not elicit epinephrine response, brain cell malfunction when deprived of glucose, cognitive behavior affected, have slurred speech, confusion, seizures, may have coma leading to death if not corrected, slow decline in blood glucose level without sudden release of epinephrine

          

  5. high serum insulin, low C-peptide levels, low proinsulin levelsindicative of reactive hypoglycaemia, sulphonylurea overdose--promote pancreatic secretion of insulin