When the patient becomes normotensive, this solution is often used to provide both electrolytes and water for normal excretion of metabolic wastes.
acute tubular necrosis
Prolonged FVD may cause a serious problem with renal function. The HCP needs to see if oliguria is produced from FVD or more seriously, produced by _______ ______ _______ from prolonged FVD. The fluid challenge test will verify this problem.
Fluid Volume deficit should be monitored at least every ___ hours. Shock can occur if 25% or more of the intravascular volume is depleted.
Symptoms associated with FVD
a decrease in body temperature, loss of a pound roughly represents 500mL of fluid loss. weak rapid pulse, orthostatic hypotension, cold extremities, declined mental function, additional longitudinal furrows on the tongue and tongue is smaller, low central venous pressure.
always secondary to an increased total body sodium content.
Symptoms of hypervolemia
edema, distended neck veins, crackles, tachycardia, hypertension, increased pulse pressure, increased central venous pressure, increased weight, increased urine, shortness of breath or wheezing.
Hypervolemic lab values
low BUN and hematocrit levels
occurs when aldosterone is chronically stimulated and the sodium level in urine does not rise
block 5% to 10% of total sodium reabsorption by blocking the distal tubule
block 20 to 30% of total sodium reabsorption by blocking sodium reabsorption in the loop of henle
electrolyte to watch when administering diuretics because hypo or hyperkalemia may occur, depending on where the diuretics work in the kidney. (Hyperkalemia can only occur if diuretics are used in the last distal tubule, ie. spironolectone)