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5 Written questions

5 Matching questions

  1. pulmonary embolism
  2. emphysema
  3. hypercapnia
  4. bronchiolitis
  5. hypocapnia
  1. a permanent enlargement of gas exchange airways with destruction of alveolar walls
  2. b increased amount of CO2 in the blood: depression of respiratory center by drugs; diseases of the medulla;
  3. c inflammatory obstruction
  4. d decreased amount of CO2 in the blood resulting from respiratory alkalosis
  5. e occlusion of the pulmonary vascular ed by emboli, can cause lung infarction

5 Multiple choice questions

  1. inflammatory response results in bronchial smooth musc spasm, vascular congestion, edema, production of thick mucous, impaired mucociliary function, thickening of airway walls and hyperresponsiveness; toxic neuropeptides cause acetylcholine-mediated bronchospasm
  2. bluish discoloration of skin and mucous membranes; reduced hemoglobin in blood
  3. autosomal recessive inherited disease on chromosome 7; causes abnormal expression of a protein affecting airway, bile duts, pancreas, sweat ducts and vas deferens
  4. usually viral laryngealtracheobronchitis, edema and mucous cause airway to narrow, barking cough
  5. inadequate gas exchange where either oxygen is too low or carbon dioxide is too high

5 True/False questions

  1. acute bronchitisinfection of airway usually caused by virus; manifests like pneumonia with fever, chills, cough

          

  2. aspirationfluid of solids that have gotten into the lungs

          

  3. hypoventilationinadequate alveolar ventilation inelation to metabolic demands; CO2 removal does not keep up with production and level increases in arterial blood causing hypercapnia

          

  4. hypoxemiadecreased amount of CO2 in the blood resulting from respiratory alkalosis

          

  5. pleural effusionfluid of solids that have gotten into the lungs