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5 Written questions

5 Matching questions

  1. chronic pancreatitis
  2. phases of hepatitis: icteric
  3. pancreatitis risk
  4. hepatitis s/s
  5. effects of liver pathologies
  1. a EtOH
    biliary blockage
  2. b jaundice phase; 2-6 wks
    s/s: jaundice, dark urine, pruritis, clay-colored stools (if obstruction)
    severe => hepatomegaly (tender/painful), s/s of inc estrogen/androgens, CNS changes
  3. c altered CHO, fat, protein (dec COP, dec Ig) metabolism
    altered vitamin/mineral (ADEK, some Bs, iron)
    altered clotting, absorption, manufacturing
    altered bilirubin metabolism, altered detox
    altered lymph formation
  4. d acute: mild with few to no s/s
    severe => liver necrosis/failure => life threatening
    usually regenerate but may lead to cirrhosis and chronic liver problem
  5. e DM and cancer of pancreas

5 Multiple choice questions

  1. toxins (EtOH abuse + malnutrition, drugs)
    biliary obstruction
    autoimmune, post-necrotic viral hepatitis syndrome (Hep B/C)
    metabolic defects
  2. severe liver dz (cirrhosis, hepatitis) => alters microvascular structure of liver
    inc BP in portal venous system => >10mmHg
  3. hepatocyte/canal damage
    if severe: only inc unconjugated (indirect) bilirubin
    if not severe: inc conjugated (direct) bilirubin
  4. rapid breakdown of RBCs => exceeds ability of liver to conjugate
    *inc unconjugated (indirect) bilirubin)
    *jaundice skin and sclera, pruritus
  5. gallstone formation (mostly cholesterol)

5 True/False questions

  1. cirrhosis: dec CHO, protein, fat metabolism=> dec BS, ascites


  2. hepatorenal syndromeascites
    hypotension due to liver disease can lead to ATN (acute tubular necrosis) & oliguria
    altered Na concentration
    azotemia (inc BUN and inc creatinine)


  3. choledocholithiasisstones in common bile duct


  4. insoluble bilirubinindirect or unconjugated bilirubin


  5. toxic hepatitis riskshigher risk if older, have liver dz, drink EtOH, femal, genetic liver enz defects