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5 Written questions

5 Matching questions

  1. risk for gallbladder dz
  2. #1 cause for liver transplants
  3. phases of hepatitis: prodromal
  4. cirrhosis: dec estrogen/androgen metabolism
  5. bilirubin metabolism
  1. a highly infectious 2 wks after exposure to onset of jaundice
    s/s is vague: malaise, fatigue, nausea, low-grade fever, sudden distaste for smoking/foods, joint pain
  2. b RBC destruction by macrophage => heme + globin

    globin => amino acid pool
    heme => iron => transferrin => ferritin

    heme => biliverdin
  3. c HBV and HCV
    #2 cause is EtOH
  4. d dec hair, gynecomastia, altered menses, angiomas, palmar erythema from INC HORMONES

    dec metab ADH, aldosterone => inc ADH/aldosterone
  5. e obesity, middle age, female, familial

5 Multiple choice questions

  1. severe pain RUQ abd
    N/V
    fever
    inc WBC
    inc bilirubin
    inc alkaline phospatase
  2. HBsAg (surface) is 1st marker (incubation, infectious as long as present, >6 mos = carrier or chronic
    HBeAg = highly infectious
    inc anti-HBe @ peak s/s => seroconverted
    inc anti-HBc: may be only marker, window period
    inc anti-HBs => recovery, immunity
    acute: inc IgM anti-HBc
  3. altered CHO, fat, protein (dec COP, dec Ig) metabolism
    altered vitamin/mineral (ADEK, some Bs, iron)
    altered clotting, absorption, manufacturing
    altered bilirubin metabolism, altered detox
    altered lymph formation
  4. 13 years = fibrosis
    20 years = cirrhosis
    30 years = cancer
  5. => dec BS, ascites

5 True/False questions

  1. hepatitis Atransmission: sexual, parental/body fluids, vertical (placental)
    HBsAg, HBcAg, HBeAg
    anti-HBs, anti-HBc, anti-HBe
    5-10% chronicity
    60 days incubation

          

  2. hepatorenal syndromeascites
    hypotension due to liver disease can lead to ATN (acute tubular necrosis) & oliguria
    altered Na concentration
    azotemia (inc BUN and inc creatinine)

          

  3. portal HTN etiologysevere liver dz (cirrhosis, hepatitis) => alters microvascular structure of liver
    inc BP in portal venous system => >10mmHg

          

  4. increase liver enzyems for liver pathologyaltered CHO, fat, protein (dec COP, dec Ig) metabolism
    altered vitamin/mineral (ADEK, some Bs, iron)
    altered clotting, absorption, manufacturing
    altered bilirubin metabolism, altered detox
    altered lymph formation

          

  5. cirrhosis pathologytoxins (EtOH abuse + malnutrition, drugs)
    biliary obstruction
    autoimmune, post-necrotic viral hepatitis syndrome (Hep B/C)
    metabolic defects