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5 Written questions

5 Matching questions

  1. fibrosis, cirrhosis, cancer in hepatitis C
  2. chronic pancreatitis
  3. cholestatic obstructive syndrome locations
  4. hepatitis A
  5. serology of hepatitis B
  1. a in gallbladder, common bile duct, common bile duct and pancreatic duct
  2. b transmission: FECAL-ORAL
    hep A antigen
    anti-HAV antibody
    NO chronicity
    30 days incubation
  3. c HBsAg (surface) is 1st marker (incubation, infectious as long as present, >6 mos = carrier or chronic
    HBeAg = highly infectious
    inc anti-HBe @ peak s/s => seroconverted
    inc anti-HBc: may be only marker, window period
    inc anti-HBs => recovery, immunity
    acute: inc IgM anti-HBc
  4. d 13 years = fibrosis
    20 years = cirrhosis
    30 years = cancer
  5. e DM and cancer of pancreas

5 Multiple choice questions

  1. hepatocyte/canal damage
    if severe: only inc unconjugated (indirect) bilirubin
    if not severe: inc conjugated (direct) bilirubin
  2. liver inflammation/necrosis =>
    dec bilirubin metabolism
    dec estrogen/androgen metabolism
    dec CHO, protein, fat metabolism
  3. transmission: sexual, parental/body fluids, vertical (placental)
    HBsAg, HBcAg, HBeAg
    anti-HBs, anti-HBc, anti-HBe
    5-10% chronicity
    60 days incubation
  4. direct or conjugated bilirubin
    liver has acted on
  5. portal HTN => dec liver metab => inc ammonia/toxin => blood and BBB => GIB byproducts => irritability, convulsions, coma

5 True/False questions

  1. choledocholithiasisstones in common bile duct


  2. cirrhosis: dec estrogen/androgen metabolism=> jaundice, clay stools/dk UA bleeding


  3. hepatitis pathologyinflam of liver => varying severity of hepatic cell damage => kupffer cell hyperplasia => infiltration of phagocytes to necrosis and scarring => alters microvasculature => portal HTN => liver failure and death


  4. effects of liver pathologiesaltered CHO, fat, protein (dec COP, dec Ig) metabolism
    altered vitamin/mineral (ADEK, some Bs, iron)
    altered clotting, absorption, manufacturing
    altered bilirubin metabolism, altered detox
    altered lymph formation


  5. cirrhosis etiologiestoxins (EtOH abuse + malnutrition, drugs)
    biliary obstruction
    autoimmune, post-necrotic viral hepatitis syndrome (Hep B/C)
    metabolic defects