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Clinical Syndrome of AKI/ARF
SUDDEN decrease in GFR
Inability to maintain electrolyte balance, acid-base
Normal Out Equals Normal Kidney Function?
Not necessarily. Normal output can mean kidneys are still failing
Creatinine multiplier for risk of AKI
Creatinine multiplier for Injury from AKI
Creatinine multiplier for Failure from AKI
1.5x normal / UO ≤5mL per kilo per hour x6hours
2x normal / UO ≤5mL per kilo per hour x12hours
3x normal / UO ≤.3mL per kilo per hour x24hours
Intra-Renal Failure Caused By?
Sodium ∧∨ During Oligeric Phase?
Body becomes HYPONatremic because the kidney cannot conserve sodium. (Possible Cerebral Edema)
Potassium Level During AKI?
Hyperkalemia + Even More HyperKalemia due to excess H+ ions kicking K+ out of cells.
Evidence of NeuroLogic Problem d/t Ammonia metabolism
Asterixis. Flapping tremor usually seen with Liver Disease
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