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Rado quiz 2: 2nd half
Terms in this set (40)
Localized hypoplastic enamel defect of the crown of a permanent tooth. An extension of periapical infection from the overlying primary tooth and/or trauma during crown formation. Ameloblasts are disrupted
generalized developmental disturbance that interferes with normal enamel formation. May be AD, AR or x-linked. Dentin and root are formed normally. There are 4 types
4 types of amelogensis imperfecta
hypoplasitc, hypomaturation, hypocalcification, hypomaturation and hypo calcification combined
type of amelogenesis imperfecta where teeth have very thin enamel with visible show of yellow- brown dentin. Teeth may be pitted and rough or smooth and glossy. Loss of proximal contacts due to square crown shape.
type of amelogensis imperfecta where there is normal enamel thickness but the enamel is not as hard. teeth have a molted appearance and enamel can flake and crack away from the dentin
type of amelogensis imperfecta where the teeth are normal size and shape but they are less mineralized. Enamel fractures easily and is less dense than dentin.
incomplete or improper development of dentin tissue. teeth have a translucent appearance. Crowns are easily fractured and severe wear is common. 3 types.
type of dentinogenesis imperfecta that is associated with osteogenesis imperfecta
dentinogenesis imperfecta type that only affects dentin. is the MOST COMMON type
dentinogenesis imperfecta type that is rare. Multiple pulp exposures and often see "shell teeth".
partial or complete obliteration of pulp chambers, cervical constriction of tooth (bulbous crowns), short and slender roots
radiographic features of dentinogenesis imperfecta
faulty development of dentin two types.
dentin dysplasia type that deals with ROOTS. Short roots, conical in shape. Can be known as "rootless teeth"
dentin dysplasia that deals with the CROWNS of teeth. normal roots. Flame/thistle/chevron shaped pulp canals
localized disturbance of both dentin and enamel. Large pulp chambers and thin enamel. Hypocalcified and thin dentin. Usually restricted to a region (like a single quadrant), also known as "ghost teeth"
Globule of enamel that occurs on the roots of molars
(only in like 3% of the population)
Predisposes patient to periodontal pockets. 1-3mm in diameter
hypoplasia of permanent incisors (hutchinson's incisors) and/or 1st molars (mulberry molars). Affects 30% of people with congenital syphilis. Spirochetes are the cause.
4 terms for variations in position
impaction, transposition, ankylosis, ectopia
These include: TMJ abnormalities, cleft palate, maxillary sinus anomalies, craniofacial development disturbances
lingual salivary gland depression, stafne bone cyst, latent bone cyst are all examples of what?
developmental salivary gland defects
most common salivary gland defect. Caused by an ingrowth of the salivary gland. Looks like a well defined ovoid radiolucency with cortical border, found below the IAN and anterior to the angle of the mandible. in the PM area
submandibular gland fossa
salivary gland defect seen more in males; well demarcated radiolucency of the mandible secondary to invagination of the lingual surface of the jaw
located in the posterior mandible below the mandibular canal
stafne bone defect
small localized compact bone growths noted most commonly on facial surface of alveolar process of maxilla
which tori is more common? Mx or md?
internal counterparts to exostoses
No condyle! There will be no angle of the mandible since the condyle isn't present to produce it
condyle with 2 heads
well circumscribed, radiolucency between the maxillary centrals
incisive canal cyst
expansion of the maxillary sinus into edentulous spaces
small maxillary sinus, can cause patients to develop chronic sinus infections because of the smaller sinus
sinus never forms
clavicles (hypoplasia) and skull (large head with frontal and parietal bossing) affected. hypertelorism, delayed closure of fontanels and sutures with multiple wormian bones, high arched palate. DENTAL FINDINGS: multiple malformed and supernumerary teeth (up to 60), retention of primary teeth and delayed eruption, course trabeculation and dense bone, narrow mandibular rami, slender and pointed condyles
cleidocranial dypslasia (dysostosis)
hypertolerism, shallow orbits, prominent mandible, maxillary hyperplasia, partially obstructed nasal passages, narrow palate, early closure of cranial sutures, frontal bossing, "beaten metal appearance"
mandibular dysostosis. Missing or hypoplasia of zygomatic bones, small and narrow face, wide and downturned mouth (short mandibular rami, antegonial notching, reverse bowing of horizontal rami), malformation of the ear (deafness), 1/3 have cleft palate and high arching, class II open bite, small paranasal sinuses
treacher collins syndrome
unusual growth of half of the face. Mental deficiency, skin abnormalities, scoliosis, GI tract abnormalities. Associated with different tumors
enlargement of the mandible, maxilla, zygoma and frontal bones on the affected side. Teeth unilaterally enlarged and accelerated in development, unilateral tongue and alveolar bone enlargement. Premature loss of primary teeth on affected side
Unilateral delayed eruption, hypodontia, malocclusion, midline of face turned toward affected side and occlusal plane canted to the affected side. May see underdeveloped or missing condyle/coronoid process/ or rams. Flat zygomatic arch without glenoid fossa
the failure of the palate to close during the early development of the fetus (lateral palatal shelves fail to fuse); can be unilateral or bilateral.
Dental: can see missing, hypoplstidc or supernumerary teeth
Radiographic: well defined vertical radiolucent defect. missing maxillary lateral incisors, supernumerary teeth, developmental tooth delay, hypodontia
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