asymptomatic but higher glucose readingsSecond fasting glucose should be done when?glucose control
Insulin therapy
Exercise
Nutrition
Social supportTx focuses for diabetesDoctors
Diabetic nurse educator
Social work
DietitianTx team for diabetesInsulin
Nutritional therapy—carb counting
BS monitoring 4-8 times day
Regular exercise—>increases insulin sensitivityWhat does Life long treatment of diabetes consist of?every 3 monthsHow often is an A1C done during insulin therapy?less than 7.5%What is the goal A1C during insulin therapy?Stress
Infection
IllnessFactors that can cause an acute change in insulin needs?administered once per day using a very long acting insulin
(Glargine/Detemir)Basal insulin therapyrapid acting insulin administered with each meal or snack based on carb count and blood glucose level
(Lispro/glulisine/aspart)Bolus insulin therapybasal-bolus therapyWhat is the recommended insulin therapy from the American Diabetic Association?decrease insulin—>increase blood glucose—> decreases cell glucose
Cells look to free fatty acids when they don't have glucose
—> leads to keto productionWhat is occurring in diabetes 1?rapid onset
repeating something over and over
pallor
sweating
hunger
double vision
tremors
seizures
numb lips/mouthHypoglycemia s/sgradual onset
lethargy
sleepy
slowed response
rapid/deep breathing
flushed dry skin
blurred vision
Fatigue
shockHyperglycemia s/sIncorrect insulin dosage
Missed insulin dosage
Incorrect administration of insulin
Surgery
Trauma
IllnessDKA occurs when?3 poly's present
Abd or chest pain
Nausea & vomiting
Hypotension
Dehydration
Weight loss
Tachycardia
Flushed ears & cheeks
Kussmaul respirations
Fruity breath
Altered LOC
Hyperglycemia, glycosuria, ketonurias/s of DKAD/t metabolic acidosisWhy does a DKA patient present with abd/chest pain, N/V, and Kussmaul RespirationsIsotonic fluid with electrolytes--dehydration
Short acting insulin Pump
Potassium ReplacementTX of DKAdecreasing serum glucose should not be at a rate faster than 100mg/dl/hr
Risk for cerebral edema
Insulin binds to IV tubing-- run 50-100 ml prior to administration
K+ shifts into cells-->hypokalemiaNursing considerations for IV insulinCerebral edemawhat is the most common complication for children with DKA and the most common cause of death in children with diabetes?oral feeds
SQ insulin
Labs for hypokalemia
CR monitorAfter a DKA patient is stable how are they progressed?Vague, long-standing symptoms develop gradually:
Obesity
Glucose in urine with or without ketones
Ketoacidosis possible
Hypertension
Androgen mediated problems
Excessive weight gain and fatigueType 2 DMAcne
hirsutism
Menstral disturbances
Polycystic ovary disease
Blurred visionAndrogen mediated problems in type 2 DMHyperpigmentation
Thickening skin
- Neck Armpit GroinAcanthosis nigricansFalse
only type 2T/F acanthosis nigricans is found in DM 1+2True
very mild if present in type 2T/F the 3P's are primarily seen with type 1 diabetesObesity
Acanthosis nigricans
Glucose levels above 200 mg/dl without fasting
Fasting glucose above 126 mg/dl
High blood pressure
Possible dyslipidemia
Might use A1CDX testing for DM2Normalize blood glucose and HbA1C
Decreasing weight
Increasing exercise
Normalize lipid profile and BP
MetforminTX of DM2True
may need insulin to stabilizeT/F A type 2 Diabetic can present with DKA