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What is the major stimulus for the release of parathyroid hormone from the parathyroid gland
The venous conncection between the hypothalamus and the adenohophysis is called the
Hypophyseal Portal System
Non hormonal material secreted through dicts onto internal and external body surfaces
The most common mechanism for hormonal activity on target cells
Hormones stimulate messenger activation or deactivation which affect other cell events
Bind to intracellular receptors within hte cell nucleus and stimulate gene activation
Bind to specific receptros on the surface of the cell and function by way of a second messenger system
Amino Acid hormones
Enzyme link between a first messenger and a second messenger in a cell activated by an amino acid hormone
When stimulated by FSH, follicle cells in the ovary produce large quantities of which femal hormone
Produced in heart cells, promotes sodium loss, suppresses ADH secretion, reduces blood pressure by decreasing blood volume
Atrial Natriuretic Peptide Hormone
Hormone produced in the kidneys that increases the number of red blood cells released from bone marrow
Caused by excess secretion of growth hormone prior to puberty, body features are normal but very large
Hormone secretion that would be affected by damage to the superotpic nucleus of the hypothalamus
Damage to the cells of the zona fasciculate of the adrenal cortex would result in
decreased glcogenesis and the ability to convert lipids to glucose
A rise in angiotensin 2 levels would result in
Fluid retention because of angiotensin stimulating release of aldosterone
ductless gland, through which hormones are released into the blood stream and effects other glands
Hormone secreted by the parathyroid gland responsible for increasing plasma calcium levels
an autoimmune disease where the body produces antibodies which act like TSH and leads to increased basal metabloic rate, weight loss, sweating, etc.
Physiologic doses of this hormone cause gluconeognesis. Pharmacologic doses depress the immune system
the condition caused by excessive clinical adminstration of cortisone or glucocorticoids characterized by persistent hyperglycemia, losses of muscle and bone protein, water and salt retention, resulting in mooned face
Hormones secreted in direct response to changing blood levels of specific ions and chemicals
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