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Study of disease inclucing fuctional, biochemical and structural (morphological) alterations, causes of disease, and sequence of events in disease
Postmortem examination of the body to determine the nature of pathological processes that contribute to death or disease
Abnormal body process with characteristic signs which begins at the molecular and cellular level and may effect all or part of the body
Mechanism of disease development from initial stimulus to ultimate expression in response of cell/tissue to the etiology
Molecular & Morphologic Changes
Biochemical and structural alterations induced in the cells and organs of the body
Concise statement or conclusion concerning the nature, cause or name of a disease or a disease process
Clinical Pathologic Diagnosis
Diagnosis based on changes observed in the chemistry of fluids and the hematology, structure and function of cells collected from the living patient
Early, sub-lethal manifestation of cell damage, characterized by increased cell size and volume
Early, sub-lethal cell damage characterized by intracytoplasmic intracytoplasmic vacuolation due to the presence of vacuoles of fat
Cellular or tissue death in a living organism characterized by preservation of tissue architecture and cellular outline; Most common manifestation of cell death
A variant of coagulation necrosis characterized by loss of tissue architecture and cellular detail
Dry Gangrenous Necrosis
Form of coagulation necrosis follow by dehydration of affected area and mummification
Moist/Wet Gangrenous Necrosis
Type of necrosis modified by the liquefactive action of saprophytic bacteria causing putrefaction
Gas Gangrenous Necrosis
Type of necrosis characterized by production of gas bubbles in the necrotic tissue by invading bacteria
Individual cell death as a result of activation of a genetically programmed cell death pathway
Increase in the size of a tissue or organ due to an increase in cell size without an increase in the number of cells
Increase in organ size or tissue mass caused by an increase in the number of constituent cells
Reversible change in which one differentiated cell type is replace by another differentiated cell type
Accumulates in neurons, cardiac myocytes, thyroid epithelium, hepatocytes, with progressive increase with age and after chronic injury
Brown, amorphous, granular, iron-containing pigment formed as a result of lysis of erythrocytes
Deposition of air pollutants such as coal dust, carbon particles into the lungs and local LN
Deposition of asbestos into lung producing chronic lung injury and associates with neoplasm
Red staining of tissues due to postmortem lysis of RBCs with resulting release of hemoglobin
Leakage of bile from the gall bladder and major bile ducts which stains adjacent tisses green to yellow
Describes an artifactual black discoloration of tissues (similar in appearance to melanosis)
Depends on body temperature before death, size of body, fat stores, hair covering, environmental temperature, etc
The enzymatic decomposition of organic materil with production of foul-smelling compounds
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