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30 terms

comd 442 midterm

STUDY
PLAY
anotia
absence of pinna
atresia
closing off of ear canal
stenosis
narrowing of EAC
microtia
small ear/pinna
supernumerary hillocks
ear tags
aural discharge
ear wax, clear fluid-trauma, cloudy fluid, blood, foreign bodies
bony growths
exotoses (multiple growths), osteoma (single growth)
inflammatory conditions
otitis externa, perichondritis (wrestler's ear), furuncle (pimple)
conductive system best assessed
visually, tympanometry
tympanometry measures
tympanic membrane mobility, ME pressure, static compliance (mass, friction, stiffness), patency of PE tubes, ear canal volume
static compliance
mass - ossicles, friction - muscles and ligaments, stiffness - footplate of stapes
anamolies of ME occur with
atresia, cleft palate, microtia; often corrected by surgery
anamolies of ME include
abnormalities, fusion/absence of ossicles
malformations of the inner ear
michel aplasia (inner ear/auditory nerve fail to fully develop), mondini aplasia (incomplete development of inner/middle/outer ear), scheibe aplasia (most common, membranous portion of cochlea-stria vascularis-organ of corti)
meningitis
hearing loss noted during first 2 weeks of illness, sensorineural loss, associated with upper respiratory infections, diagnose quickly, watch carefully-->cochlear implant possible, ABR/behavioral testing
toxoplasmosis
rare, fatal, undercooked meat, premature birth, CPalsy, blindness, hydrocephaly, appear normal at birth but symptoms appear later on; 2-7/1000 women affected, 30-40% the fetus infected as well
Other.... syphilis
STD, CNS abnormalities, sensorineural hearing loss; by age 2 hearing loss shows
Rubella
viral, 50% of time hearing loss occurs, may cause heart disease, eye problems; immunizations key
CMV - cytomegalovirus
most common, harmless to most ppl, causes vision problems, seizures, death, dev/cog. delays, transmitted through breast milk; up to 10-40% of children are carriers; no known treatment
Herpes simplex virus
genital herpes/STD, mother's first outbreak during pregnancy, sensorineural loss, 50% mortality rate
ototoxicity
drugs that cause damage, but save life (aminoglycosides, chemotherapy drugs, aspirin/quinine, loop diuretics(if with other ototoxicity drugs will cause problem)
presence of OAEs
little or no conductive loss from ME abnormality; normal OHC function; with sensorineural loss = disorder is retrocochlear
absence of OAEs
with conductive loss = does not eliminate possibility of cochlear/retrocochlear loss; with sensorineural loss = confirms cochlear pathology, but doesn't rule out retrocochlear
assessment of neural system
measures of temporal and processing aspect; brain imaging; AEPs
Types of AEPs
ECoG, ABR, AMLR, ALR, P300
ECoG
patients sleeping, SP(IHCs), AP(distal portion of 8th nerve, IHC-Spiral ganglion), CM(OHCs); 1.5-2ms
ABR
2-10ms; 8th nerve; screening
AMLR
12-60ms; auditory thalamus, primary auditory cortex; awake
ALR
75-200ms, awake, auditory cortex
P300
200-600ms, APD, awake, medial temporal lobe