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Which adrenoceptor?Vasoconstriction, increased TPR, increased BP, mydriasis, increased closure of internal sphincter of the bladder
Which adrenoceptor? Inhibition of NE release, inhibition of ACh release, inhibition of insulin release?
Which adrenoceptor? Tachycardia, increased lipolysis, increased myocardial contractility, increased renin release
Which adrenoceptor? Vasodilation, slightly decreased peripheral resistance, bronchodilation, increased release of glucagon, increased muscle and liver glycogenolysis
How does epi affect cardiovascular?
Positive inotropic (contractility) and rate (chronotropic) via Beta 1.
How does NE affect peripheral resistance?
Constricts all blood vessels, causing increased peripheral resistance
How does isoproterenol affect systolic and diastolic pressure?
Moderate increase in systolic, markedly decrease in diastolic pressure.
How does isoproterenol affect peripheral resistance?
Significant decrease (hence decrease in diastolic pressure)
Raises blood pressure by stimulating Beta 1 and alpha 1 vasoconstriction, but also enhances perfusion to the kidney and splanchnic areas.
What is dobutamine used for? What receptor?
Increase cardiac output in congestive heart failure. Beta-1
Dobutamine should be used with caution in what types of patients? Why?
Atrial fibrillation patients. Drug increases atrioventricular conduction.
Nasal decongestant, terminate episodes of supraventricular tachyardia.
Phenylephrine (alpha-1 agonist)
What drug can be used to increase cardiac output without significant increase in heart rate?
Dobutamine (only beta 1)
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