Major infarction in the territory of the superior division causes a dense sensorimotor deficit in the contralateral face, arm, but, to a lesser extent the leg, as well as ipsilateral deviation of the head and eyes; i.e., it mimics the syndrome of MCA stem occlusion except that the leg and foot are partly spared. With left-sided lesions there is initially a global aphasia, which changes to a predominantly nonfluent (Broca's) aphasia, with the emergence of an effortful, hesitant, grammatically simplified, and dysmelodic speech The usual result in left-sided lesions is a Wernicke's aphasia, which generally remains static for days or weeks after which some improvement can be expected. In less-extensive infarcts that are the result of selective distal branch occlusions (superior parietal, angular, or posterior temporal), the deficit in comprehension of spoken and written language may be especially severe. Again, after a few months, the deficits usually improve, often to the point where they are evident only in self-generated efforts to read and copy visually presented words or phrases. With either right- or left-hemispheric lesions, there is usually a superior quadrantanopia or homonymous hemianopia and, with right-sided ones, a left visual neglect and other signs of amorphosynthesis. Wallenberg syndrome. Involves V, VIII, IX, X, sympathetic, spinothalamic, and cerebellum. Decreased contralateral P+T in body, ipsilateral P+T in face, ipsilateral Horner's, nausea, vomiting and vertigo. Also decreased gag, hoarseness, dysphagia, cord paralysis, decreased taste.